Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibition.
Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the original research article "Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT1 receptor-NOX2 signaling axis" [1].
['Rajtik|Tomas|T|', 'Carnicka|Slavka|S|', 'Szobi|Adrian|A|', 'Giricz|Zoltan|Z|', 'O-Uchi|Jin|J|', 'Hassova|Veronika|V|', 'Svec|Pavel|P|', 'Ferdinandy|Peter|P|', 'Ravingerova|Tanya|T|', 'Adameova|Adriana|A|']
Myocardial ischemia/reperfusion injury Apoptosis Necrosis AT1 receptor blockade CaMKII inhibition