XXXX and signal averaged electrocardiogram.

@entity1 @entity296 @entity301 @entity27 @entity5036 @entity943
@entity1 with @entity27 demonstrate an increased rate of @entity943 . The present study was designed in order to investigate whether these @entity943 may be related to @entity296 , since @entity296 of various aetiologies has been observed to change the signal averaged electrocardiogram (ECG). Signal averaged ECG was registered in 23 @entity1 with @entity27 diagnosed by polysomnography (apnoea index 43 +/- 20 events.h-1, age 55 +/- 10 yrs). QRS duration, root mean square voltage of the last 40 ms of QRS, and low amplitude (< 40 mV) signal duration were determined from the vector magnitude of the QRS, high-pass filtered at 40 Hz. @entity1 with @entity301 or @entity5036 were excluded. No @entity1 showed an abnormal signal averaged ECG. Mean duration of the filtered QRS complex was 96 +/- 9 ms, root mean square voltage 38 +/- 18 microV and low amplitude signal duration 26 +/- 8 ms. These results were not significantly different from 14 snoring subjects with an apnoea/hypopnoea index < 10. Four @entity1 showed no @entity943 and six @entity1 had Lown III or IVa in the Holter ECG. Echocardiography revealed increased left atrial (43.7 +/- 4.1 mm) and interventricular septal diameters (11.3 +/- 1.4 mm). In conclusion, @entity27 does not generate a substrate for late potentials in the signal averaged ECG.
