@entity73 associated with XXXX .

@entity1 @entity73 @entity9303 @entity4 @entity346 @entity392 @entity1274 @entity10 @entity551 @entity2841
OBJECTIVE: To investigate the patterns and mechanisms of @entity73 associated with @entity551 . METHODS: We had consecutively recruited 16 @entity1 with @entity551 at the Neurology Center of Pusan National University Hospital for two years. Spontaneous, gaze-evoked, and positional @entity346 were recorded using 3D video-oculography in all @entity1 , and the majority of them also had pure tone audiometry and bithermal caloric tests. RESULTS: Of the 16 @entity1 , five (31.3%) reported neuro-otological symptoms along with the @entity10 while laboratory evaluation demonstrated @entity73 in ten (62.5%). Oculographic analyses documented spontaneous and/or positional @entity346 in six @entity1 (37.5%) including weak spontaneous @entity346 with positional modulation (n=4) and pure positional @entity346 (n=2). One @entity1 presented with recurrent spontaneous vertigo and @entity392 mimicking @entity9303 , and showed unidirectional horizontal and torsional @entity346 with normal head impulse tests during the attacks. Bithermal caloric tests were normal in all nine @entity1 tested. Audiometry showed unilateral (n=6) or bilateral (n=1) @entity1274 in seven (53.8%) of the 13 @entity1 tested. CONCLUSIONS: @entity551 frequently induces @entity73 . In addition to endolymphatic @entity4 and irritation of the vestibulocochlear nerve, compression or traction of the brainstem or cerebellum due to loss of @entity2841 buoyancy may be considered as a mechanism of frequent spontaneous or positional @entity346 in @entity1 with @entity551 .
