Preoperative cerebrovascular symptoms and @entity854 do not predict XXXX during carotid endarterectomy.

@entity1 @entity64 @entity2806 @entity63 @entity16 @entity854
BACKGROUND AND PURPOSE: The purpose of this prospective study was to establish (1) whether @entity1 with @entity16 scheduled for carotid endarterectomy had an increased incidence of electroencephalographic (EEG) abnormalities during awake baseline recordings, (2) whether these symptoms and @entity854 predicted @entity63 EEG pattern changes at carotid artery cross-clamp, and (3) whether there was an association between age, presence of EEG baseline abnormalities, and @entity63 pattern changes at carotid artery cross-clamp. METHODS: We reviewed the medical record of each @entity1 scheduled to undergo carotid endarterectomy and recorded the @entity1 's age and history of previous @entity16 . We then continuously monitored and analyzed 16 channels of anteroposterior bipolar EEG and two of referential derivations from at least 5 minutes before induction of anesthesia and throughout the operation. RESULTS: We completed 394 consecutive studies. @entity16 were related to @entity854 in awake @entity1 (P < .001) and to EEG asymmetries in anesthetized @entity1 (P < .001). Abnormal awake EEG findings were associated with asymmetries after anesthesia (P < .0001). Twenty-eight percent of both symptomatic (70/249) and asymptomatic (41/145) @entity1 had EEG @entity63 pattern changes at carotid artery cross-clamp. Neither @entity16 nor @entity854 were associated with age or the development of EEG @entity63 pattern changes at carotid artery cross-clamp. CONCLUSIONS: Despite the strong association between a history of @entity2806 and preoperative @entity854 in @entity1 undergoing carotid endarterectomy, @entity1 who have @entity64 or transient @entity63 events are at no greater risk of having EEG evidence of @entity2806 during carotid artery cross-clamp than @entity1 without symptoms and with normal baseline EEGs. We conclude that preoperative @entity854 in symptomatic @entity1 are not due to age or to insufficiency of regional cerebral blood flow.
