Cerebral syndromes of XXXX .

@entity1 @entity455 @entity2657 @entity66 @entity6 @entity308 @entity2318 @entity5507 @entity26 @entity3157 @entity772 @entity8193 @entity1270 @entity731 @entity4854
Three labile @entity6 @entity1 had recurring episodes of altered sensorium. Each had severe @entity66 with superimposed metabolic derangements, including @entity1270 , @entity4854 without @entity1270 , mild @entity5507 , and possibly @entity2657 . Two of the @entity1 were examined postmortem. Severe @entity2318 , basal ganglial calcification and destruction of small intracerebral vessels without evidence of large vessel @entity731 were found unexpectedly in one @entity1 , a rare occurrence in this country although recently reported from Europe. The other @entity1 had large vessel @entity731 only. The clinical expression of the @entity455 was modified by concurrent abnormalities and reflected the sum total of the complexities which coexisted. The pathophysiology of the unconscious state necessarily depends on the inciting factors. @entity772 is associated with @entity308 cerebral @entity26 consumption. Spinal fluid pH is usually maintained during @entity1270 but is sometimes lowered inadvertently during @entity3157 therapy, with resultant @entity772 . Other variables influencing the clinical expression, with or without @entity1270 , would include, among others, blood viscosity alterations, rapid decrements in blood sugar, and existing degrees of @entity8193 . The increasing life-span of the juvenile diabetics, favorably influenced by improved management and recently by hemodialysis, may uncover @entity455 heretofore rarely seen and create additional diagnostic and therapeutic enigmas.
