Changes in arterial @entity2693 following dialysis in relation to XXXX and to endothelial function.

@entity1 @entity1284 @entity1242 @entity1184 @entity3045 @entity2033 @entity740 @entity5225 @entity53099 @entity5707 @entity299 @entity65 @entity122 @entity2693
INTRODUCTION: Cardiovascular (CV) morbidity and mortality are greatly enhanced in @entity1 with @entity740 , partly due to increased arterial @entity2693 . MATERIAL AND METHOD: The study included 63 stable @entity2033 @entity1 . Stiffness parameters were evaluated by applanation tonometry before the mid-week @entity2033 sessions. Pre- @entity2033 bioimpedance parameters were measured. A phase angle <six degrees was previously reported as abnormal, reflecting extracellular @entity5225 . Fluid status was evaluated echocardiographic by measuring the inferior vena cava (IVC) diameter. Endothelium-dependent and endothelium-independent vascular reactivity were assessed by changes in @entity53099 following sublingual @entity1242 and inhaled @entity3045 . RESULTS: @entity122 directly correlated with @entity1 ' age and dialysis vintage. @entity1 with a phase angle <6  , were significantly overhydrated (larger IVC, increased ECW, and lower ICW), had stiffer arteries and greater @entity5707 ( @entity5707 ), compared with those with a phase angle >6  . Overhydration increases arterial @entity2693 , but has no influence on either EID or ED vascular reactivity. CONCLUSION: In hemodialysis, @entity1284 is an important contributor to increased arterial @entity2693 and modifies @entity65 especially by @entity1184 . Achieving normohydration may significantly ameliorate @entity299 and arterial @entity2693 and may impact major clinical events and CV mortality.
