@entity148 trait in XXXX with stress-induced cardiomyopathy: a case-control study.

@entity1 @entity101 @entity908 @entity148 @entity661 @entity299 @entity583 @entity204
BACKGROUND: Stress-induced @entity661 ( @entity661 ) is an @entity299 , mimics @entity583 ( @entity583 ), largely confined to postmenopausal @entity1 , frequently precipitated by a stressful event. The pathogenesis of @entity661 is still unknown. Some authors hypothesized a possible connection between @entity661 and @entity148 , but no previous study analyzed the relationship between @entity148 trait and @entity661 . This study sought to assess the potential role of @entity148 trait in the development and clinical course of @entity661 . METHODS: We included in the present prospective case-control study 50 consecutive @entity1 admitted to our Hospital with a diagnosis of @entity661 according to the Mayo Clinic criteria. Fifty control @entity1 with anterior @entity583 matched for clinical characteristics such as age, gender, and @entity101 were selected. During the hospitalization, all @entity1 were asked to complete the Spielberger Trait @entity148 Inventory (STAI) scale for measuring self-reported trait @entity148 (Trait-A). Outcome measures at follow-up were @entity204 , @entity661 recurrence, and rehospitalization. RESULTS: The mean value of STAI scale was 46    12 in @entity661 @entity1 and 45    14 in @entity583 @entity1 (p = 0.815). High- @entity148 trait (STAI scale value >=  40) was documented in 30 (60%) @entity661 @entity1 and in 26 (52%) @entity583 @entity1 (p = 0.387). At multivariate analysis, predictors of @entity661 were lower peak @entity908 kinase value (HR 0.999; 95% CI 0.998-0.999; p = 0.018) and an antecedent stressful trigger event (HR 45.487; 95% CI 6.471-319.759; p = 0.001), but @entity148 trait was not. There were no differences in outcome measures between @entity661 @entity1 with or without high- @entity148 trait. CONCLUSION: In @entity661 @entity1 , high- @entity148 trait is a common finding but it is not significantly more frequent than in @entity1 with @entity583 . Moreover, a high- @entity148 trait seems to be neither associated with a worse clinical outcome nor a predictor of @entity661 . Our study do not support the routine evaluation of @entity148 trait in @entity1 with @entity661 .
