Pathology of the XXXX -origin influenza A (H1N1) flu.

@entity1 @entity1707 @entity932 @entity455 @entity548 @entity63 @entity1423 @entity16 @entity3794 @entity1188 @entity715 @entity204 @entity2401
Pathological studies would aid in finding the real causes of @entity204 and in outlining adequate strategies for treatment regarding @entity1 with poor clinical outcome of influenza A H1N1 @entity1707 flu. We describe the autopsy findings of six cases of influenza A H1N1 @entity1707 flu. The lungs in these cases had an @entity3794 with hyaline membranes. Immunohistochemistry for influenza was positive only in lungs (in pneumocytes, in macrophages, in some multinucleate cells in alveoli, and in blood vessel walls) of two cases. @entity932 was observed in four of the cases and focally in one case. Focal @entity2401 was observed in one case. Coagulation @entity1188 ( @entity63 ) were observed in the pancreas of two cases and in the spleen of two cases. Our results indicate that there was marked replication of the virus in alveoli in the more recently infected cases, which could explain the extensive @entity16 . In our cases, there were important vascular phenomena that resulted in @entity548 and @entity715 , but without marked @entity1423 and coagulation cascade disruptions. This would be attributed to hemodynamic disruption. However, it is possible that the @entity548 in the brain are due to @entity455 or to an increase of endothelial permeability.
