Patterns of incidence and XXXX risk factors for traumatic events.

@entity1 @entity146 @entity1005 @entity1316 @entity148 @entity734 @entity130
Individual differences are thought to influence the propensity for exposure to @entity130 and the subsequent development of @entity1005 ( @entity1005 ) symptoms. Prior research has identified pre-existing @entity1316 as one such individual risk factor for traumatic events as well as for @entity1005 . The present study reports the incidence of traumatic events (and @entity1005 ) and examines @entity146 risk factors for @entity130 exposure in a prospective community sample. Data come from a prospective, longitudinal epidemiological study of adolescents and young adults (age 14-24) in Munich, Germany. Respondent diagnoses (N = 2,548) at baseline and at follow-up 34-50 months later were considered. Psychiatric diagnoses at baseline were examined as predictors of qualifying @entity130 . Baseline prevalence of @entity1 having experienced @entity130 meeting DSM-IV A1 and A2 criteria ('qualifying @entity130 ') was 16.7%; during the follow-up period, 20.3% @entity1 had experienced incident (new) qualifying traumata. The prevalence of @entity1005 , including subthreshold cases, at baseline was 5.6%; by the end of the follow-up period this had increased to 10.3%. Presence of an @entity148 at baseline predicted exposure to qualifying @entity130 during the follow-up period (adjusted ORs ranging from 1.36 for any @entity130 type to 3.00 for sexual @entity130 ); this association was apparently due to an increased tendency to report events as being particularly horrific (meeting A2 criteria). In contrast, presence of illicit drug use predicted the onset of traumatic events (specifically assaultive and sexual @entity130 ) meeting at least A1 criteria, suggesting an actual exposure to these types of traumatic events for this class of disorders. In this prospective study of urban adolescents and young adults, certain classes of pre-existing @entity146 (most notably @entity148 and illicit @entity734 ) were associated with increased risk for qualifying traumatic events. The mechanisms by which premorbid @entity146 promote exposure to traumatic events are unknown. Better understanding of these pathways may lead to novel strategies for primary and secondary prevention of @entity1005 .
