[On a case of @entity2026 with XXXX (author's transl)].

@entity1 @entity548 @entity75 @entity73 @entity5785 @entity2026 @entity9479 @entity74 @entity5547 @entity2459
The possibility of @entity74 caused by @entity9479 has been supported by anatomo-clinical findings, stereotaxic surgery and more recently by radiological investigations with new techniques, i.e. ct and brain scan. A case of thalamic @entity2026 investigated with these techniques and the Luria's neuropsychological test is reported in this paper. The case concerns a 50 years old @entity1 who developed a @entity2459 with marked @entity5785 . C.T. and radioisotope brain scan revealed a left capsular @entity548 with involvement of thalamic structures. Later, when this @entity1 's @entity2026 decreased, Luria's test showed reduction of spontaneous speech, @entity5547 , @entity74 , distractibility without @entity73 and repitition. These findings are similar to those reported by some AA. in @entity9479 and similar to aphasic syndromes caused by @entity75 . Another important observation was the presence of widespread cortical functional disorder involving the left fronto-temporo-parietal region without evidence of cortical anatomical alteration. The above data and those reported in the literature suggest a very close relationship between thalamic structures, other central nuclei and cortical areas particularly in the control of speech.
