XXXX : sonographic demonstration and significance in neonates.

@entity1 @entity932 @entity548 @entity1276 @entity5034 @entity281 @entity40441
In the neonate, @entity40441 is believed to be related to @entity5034 . Yet, in our experience, this sign is much more often observed in association with subacute @entity548 ( @entity548 ), without @entity281 . Sixty premature neonates were prospectively studied. The observations of transfontanellar sonograms ( @entity932 , ependymal echogenicity, and @entity1276 ) were correlated with the results of MRI, lumbar punctures and clinical work-up. @entity932 was detected in 28 @entity1 , and @entity40441 was observed in 21 of them, all of whom had @entity548 . In 9 of these 21 @entity1 @entity548 was diagnosed retrospectively thanks to the visualization of the hyperechoic ependyma. In all but one, this sign persisted for at least 2 months after disappearance of other signs of @entity548 . MRI demonstrated the presence of hemosiderin and ferritin in ependymal or subependymal location only in @entity1 with hyperechoic ependyma. One of our @entity1 had in utero diagnosis of @entity548 owing to the visualization of the same hyperechoic aspect of the ependyma. Nine of the neonates with hyperechoic ependyma developed @entity1276 , and three underwent surgery. Hyperechoic thickening of the ependyma in prematures often results from a subacute @entity548 . It is related to hemoglobin catabolites which can be detected by MRI. It does not require immediate potentially harmful diagnostic punctures. The presence of this hyperechoic rim allows a retrospective diagnosis of @entity548 and indicates a clinical and sonographic follow-up in newborns at risk for secondary @entity1276 .
