XXXX . Ultrastructural observation of a case.

@entity1 @entity932 @entity1369 @entity101 @entity1184 @entity4091 @entity16663 @entity619 @entity25010 @entity972 @entity916 @entity6392 @entity1371 @entity1482
A 56-year-old @entity1 presented with @entity932 . Laboratory examinations revealed severe @entity101 , @entity1371 , elevated @entity1369 excretion, and suppressed plasma @entity916 activity. @entity16663 was suspected and adrenalectomy was performed. The laboratory data after operation, however, showed no significant difference from the preoperative data. On the basis of further examinations, @entity619 was administered and returned blood pressure to normal, and also normalized serum @entity972 , plasma @entity1369 , and @entity916 activity. The @entity1 's illness was diagnosed as @entity25010 . Light microscopically, the @entity4091 glomerulosa was @entity1184 and the @entity4091 decreased in lipid droplets and was centrifugally arranged in small alveoli. Electronmicroscopically, the cells of the @entity4091 had several lipid droplets and well-developed sER. Mitochondria were round to oval with lamellar or lamellovesicular cristae. These findings were evidence of @entity6392 . The cytoplasm of the cells also contained @entity1482 bodies. Therefore, it is assumed that the @entity1369 , which induced the disorder, was produced mainly in the @entity4091 .
