Changes in cardiac XXXX status after cerebral ischemia.

@entity1 @entity64 @entity63 @entity999 @entity4615 @entity955 @entity299 @entity1856 @entity715 @entity1229 @entity385
BACKGROUND AND PURPOSE: Intracardiac thrombi are a potential source of @entity999 . The disposition of @entity4615 in ischemic @entity385 ( @entity385 ) @entity1 was investigated over a 10-month period using omniplanar transesophageal echocardiography (TEE). METHODS: One hundred and five @entity1 underwent TEE examination at <1 month and again at 9-12 months (mean 10 months) after symptom onset. TEEs were evaluated for thrombi in the left atrium, left @entity955 and left ventricle. @entity64 risk factors and TEE findings were compared between @entity1 with and without new thrombi on follow-up TEE. Similar comparisons were made in @entity1 with and without disappearance of thrombi on the follow-up TEE. The effect of anticoagulants was evaluated. RESULTS: Intracardiac thrombi were found initially in 18% (95% CI 11-25%) of @entity1 in 79% (95% CI 61-97%) of whom the thrombi disappeared on the follow-up TEE; this significantly correlated with warfarin anticoagulation (p = 0.037). In the remainder 82% (95% CI 75-89%) @entity1 , new thrombi were detected on the follow-up TEE in 8% (2-14%). These were older (p = 0.009), and not on anticoagulation. @entity1 with aortic @entity1856 >/=4 mm were also more likely to develop new @entity4615 (p = 0.001). CONCLUSIONS: Anticoagulation with @entity1229 for 10 months is associated with conditions leading to disappearance of @entity4615 after a cerebral @entity63 event and hence has a probable therapeutic role. Older patients, not anticoagulated, and those with significant aortic @entity1856 >/=4 mm may have increased probability for de novo @entity715 development. These @entity1 may be at risk of @entity299 and could be considered for prophylactic anticoagulation.
