[The effect of smoking on resting energy expenditure in XXXX with early @entity2316 ].

@entity1 @entity32 @entity1865 @entity2316 @entity310 @entity1234 @entity1254 @entity224 @entity1386 @entity1851
OBJECTIVE: To study the effect of smoking on resting energy expenditure (REE) and the relationships among REE, smoking, @entity32 and oxidative stress in @entity1 with @entity2316 . METHODS: A case control study of 31 smokers and 40 non-smokers with early stage of @entity2316 (stage III) were performed to evaluate the chronic effect of smoking on REE. @entity1386 ( @entity1386 ), biomarkers of oxidative stress @entity1234 ( @entity1865 ), @entity224 dismutase (SOD) and @entity32 high-sensitivity @entity1851 (hs-CRP), @entity1254 , @entity310 were also measured in these subjects. Data were analyzed by Pearson correlation analysis. RESULTS: Compared with non-smokers, REE/ @entity1386 in smokers group was significantly increased by 15.96% (P = 0.001). Pearson analysis showed that smoking was significantly correlated with REE/ @entity1386 (t = 0.395, P = 0.001). There were significantly different between smokers and non-smokers in @entity1865 , SOD and hs-CRP (P < 0.05). But no difference between two groups in @entity1254 and @entity310 (P > 0.05). No significant relationships between REE/ @entity1386 and @entity1865 , SOD, hs-CRP, @entity1254 , @entity310 was found (P > 0.05). CONCLUSION: Chronic smoking can lead to increased REE, arouse oxidative stress and inflammatory in @entity1 with early stage of @entity2316 . However, there is no relationship between increased REE due to smoking and oxidative stress and inflammatory.
