Reversal of @entity1985 due to reactive amyloidosis (AA-type) after excision of localized XXXX .

@entity1 @entity97 @entity650 @entity12042 @entity1851 @entity353 @entity32 @entity2783 @entity527 @entity308 @entity12692 @entity1395 @entity1985 @entity4841 @entity14756
The @entity1 (TAL), a chronic asymptomatic @entity97 carrier with HBsAg-anti-HBsAg circulating immune complexes, was admitted to our hospital because of a @entity1985 due to @entity12042 . There was no family history of @entity14756 . Recurrent @entity4841 , @entity1395 , and @entity353 were the prominent clinical features. Laboratory test results showing that severe chronic inflammatory activity had been present for 6 years. @entity650 ( @entity650 ) serum concentration was 10 times normal and @entity1851 was 1.9 mg/ml. A complex immunological picture was also present (immune complex formation, exuberant B-cell reactivity, and decrease in the number of CD4 T cells). A localized form of @entity12692 ( @entity12692 ) (plasma-cell type) was diagnosed by surgical excision of a giant axillary lymph node. AA amyloid was present in the blood vessels. Within 60 days after excision of the mass, the systemic symptoms subsided, laboratory signs of inflammatory activity disappeared and @entity650 serum concentration returned to normal, thus establishing a causal relationship between the localized @entity12692 , elevated @entity650 concentration and the @entity32 responsible for @entity2783 . At 10 months of follow-up, the @entity1985 has reversed, kidney function has slowly ameliorated, and the @entity1 has gained 12 kg. Abdominal fat aspirates drawn to search for amyloid, positive before surgery, were subsequently negative. The latter finding, and the remission of the @entity1985 , provided strong evidence for regression of the @entity527 . However, the HBsAg-anti-HBsAg immune complexes and @entity308 of T-helper cell activity persist. This immunological derangement is therefore not a consequence of @entity12692 . Chronic stimulation of the immune system due to the @entity1 's inability to eliminate @entity97 , in the contest of perturbed immunity, may have favored the genesis of the lymphadenopathy.
