A reversible type of XXXX following @entity63 in the gerbil hippocampus.

@entity4485 @entity1399 @entity63 @entity432 @entity528
The Mongolian gerbil is known to develop delayed @entity528 in the hippocampus following brief @entity63 (Brain Res 239: 57-69, 1982). The effect of @entity4485 on this slow process of @entity528 was examined. Immediately following 5 min of bilateral @entity432 , @entity4485 (10, 20, or 40 mg/kg) was injected. The control animals received saline injection. Seven days following ischemic insult, animals were perfusion-fixed and the neuronal density in the hippocampal @entity1399 subfield was counted. Most of the neurons in the @entity1399 sector survived ischemic insult when @entity4485 was given, whereas most of control group neurons were lost without the treatment. The average neuronal density of 20 mg/kg group was 168.2 +/- 12.3 (SEM) per 1 mm linear length of the @entity1399 subfield. The density in 40 mg/kg group was 181.1 +/- 14.9. The neuronal density in the whole control group was 34.3 +/- 5.1. The density of unoperated normal gerbils was 212.3 +/- 3.9. This result indicates that the @entity528 of "delayed @entity528 " is reversible. On the other hand, when @entity4485 was injected 1 hr following @entity63 , it showed no effect. The cell change in the @entity1399 sector, reversible at the initial stage, seems to rapidly become irreversible, while neurons still remain intact morphologically.
