The incidence and causes of @entity323 after XXXX associated with posterior urethral disruption.

@entity1 @entity323 @entity174 @entity16 @entity3832 @entity130
PURPOSE: @entity323 is a common sequel of @entity174 , particularly those associated with posterior @entity130 when it can be neurogenic or arteriogenic due to damage to the cavernous nerves or branches of the pudendal arteries. We studied erectile function of @entity1 with posterior @entity130 due to @entity174 . MATERIALS AND METHODS: @entity1 referred for posterior urethral reconstruction and strictures due to @entity174 were evaluated before reconstruction. All @entity1 underwent nocturnal penile tumescence testing, and if those results were abnormal, penile duplex ultrasound with intracavernous injection was performed. @entity1 with normal vascular function on duplex ultrasound were diagnosed with @entity323 . Those @entity1 with abnormal arterial function on duplex ultrasound underwent arteriography to further define the extent and location of @entity16 . RESULTS: The study included 25 consecutive @entity1 with @entity3832 and a mean age of 28.6 years. Of the @entity1 18 (72%) had @entity323 as demonstrated by nocturnal penile tumescence and all underwent penile duplex ultrasound. Ultrasound confirmed normal vascular response in 13 of the 18 @entity1 and they were diagnosed with probable @entity323 . The remaining 5 @entity1 (28%) with @entity323 had an abnormal arterial response, and significant arterial pathology was confirmed by arteriography. CONCLUSIONS: @entity323 is common in @entity1 with @entity174 associated with @entity130 . We believe that erectile function should be assessed and documented in such @entity1 before attempting urethroplasty. In the majority of these @entity1 @entity323 is caused by disruption of the cavernous nerves with sparing of arterial inflow.
