Neurological manifestations of XXXX from left atrial ablation.

@entity1 @entity410 @entity64 @entity146 @entity1727 @entity772 @entity887 @entity292 @entity911 @entity955 @entity299 @entity158 @entity4032
@entity4032 ( @entity4032 ) is carried out as an alternative treatment of @entity955 refractory to conservative measures. An increasingly recognized complication of @entity4032 is an @entity1727 ( @entity1727 ). Neurological manifestations usually dominate the presentation of an @entity1727 and result from @entity299 of thrombi, transgressed esophageal contents, or air. AEFs have been reported after @entity4032 in 52 @entity1 (23 @entity1 , age 35-76 years), described in 24 publications, so far. AEFs manifest clinically on the average 17 days (3-38 days) after ablation. Neurological manifestations include @entity64 (n = 30), @entity410 (n = 9), transitory-ischaemic attacks (n = 6), @entity772 (n = 6), or @entity146 (n = 5). Imaging studies of the cerebrum most frequently show multiple @entity64 or air @entity887 . The diagnosis is made upon thoracic computed tomography with contrast media. An act of swallowing, trans-esophageal echocardiography, gastroscopy, or esophageal stenting must be avoided not to enlarge the shunt. The only expedient therapy is surgical closure of the @entity292 , but even then, the prognosis is poor with a mortality of 71%. AEFs should be suspected if there is a history of @entity4032 followed by fever, @entity158 , postprandial @entity64 , @entity410 , @entity772 , or @entity911 with a latency of days to 5 weeks after ablation. Diagnostic work-up must avoid measures, which enlarge the @entity292 . Treatment is surgical exclusively.
