@entity64 after acute @entity932 in XXXX with @entity7025 : six new cases and a short literature review.

@entity1 @entity64 @entity932 @entity548 @entity75 @entity1188 @entity7025 @entity1031 @entity2553 @entity1779 @entity385
BACKGROUND: We report six new cases of @entity64 after @entity1779 in @entity1 with @entity7025 ( @entity7025 ) and analyse their clinical and radiological characteristics, together with cases reported in the literature, to deduce the mechanism of @entity385 . METHODS: Six (2%) of 246 @entity1 with @entity7025 who were admitted to our hospitals between 1993 and 2009 suffered @entity385 after @entity932 . Ten @entity1 identified in the literature with the PubMed search engine were also included in this study. All the @entity75 in these 16 @entity1 were analysed according to their location, size, and number and were compared according to the spatial relationship between the @entity548 and @entity1188 , as follows: (1) anterior vs posterior involvement, (2) cortical vs subcortical involvement, (3) watershed vs non-watershed @entity1188 , (4) small vs large @entity1188 , (5) single vs multiple @entity1188 , and (6) adjacent vs distant involvement. RESULTS: Acute synchronous multiple brain @entity1188 occurred in six (38%) @entity1 and recurrent @entity1188 in three @entity1 (19%). @entity385 had mainly cortical (72%), anterior (66%), and distant involvement (75%) and were large (69%) and non-watershed (66%). @entity1188 had significantly anterior involvement (P   <   0.05), and distant @entity1188 tended to have cortical involvement. Non-watershed @entity1188 had significantly cortical involvement (P   <   0.05). @entity1188 tended to be large. @entity2553 was confirmed either pathologically or angiographically in two @entity1 with large @entity385 . CONCLUSIONS: We suggest that @entity1031 or @entity2553 plays a crucial role in the pathogenesis of @entity385 after acute @entity932 in @entity1 with @entity7025 .
