[Clinical picture and pathophysiology of XXXX ].

@entity1 @entity129 @entity1032 @entity2657 @entity428 @entity75 @entity4 @entity772 @entity1620 @entity21
In 48 @entity1 who had @entity428 (icp) this was monitored continuously by an epidural route. Icp-values were correlated with other important parameters like the mean arterial pressure (map) and the early components of brainstem acoustic evoked responses (BAER). The results were as follows: 1. focal and multifocal @entity75 resulting in @entity2657 have better prognosis than those morbid conditions causing cytotoxic @entity4 . In the letter group the whole brain's metabolism is impaired, brainstem, cerebellum and spinal cord included. 2. by monitoring map and icp we are enabled to assess the cerebral perfusion pressure. If this value falls short of 20 Torr the duration of unconsciousness and the frequency of brainstem related symptoms increases as well as @entity129 and @entity21 and @entity1032 in the long term course. 3. @entity75 and @entity75 in general are not related to icp-increase. They resemble local @entity75 , e.g. after contusions. 4. serial registration of BAER is a good aid in @entity772 . If there are critical values of the perfusion pressure mainly the waves III and V of BAER have increased peak latencies and go flattened. This trend takes place slowly within hours or even days. Loss of the components III and V is prompted by an irreversible damage of the caudal brainstem. This finding after cytotoxic brain @entity4 is even more relevant than after vasogenic @entity4 . If the component loss III-V is bilateral dissociated @entity1620 has to be anticipated. Some important issues of icp-pathophysiology are discussed according to the literature.
