Is ischemia involved in the pathogenesis of XXXX ?

@entity1 @entity450 @entity2704 @entity4185 @entity10
Regional cerebral blood flow (rCBF) was measured in 254 areas of a hemisphere with the intraarterial 133-xenon method in seven @entity1 before and during classical @entity2704 attacks. During prodromes/accompaniments focal flow was markedly reduced (mean 36 p. cent. range 22-60 p. cents). Initial focal @entity450 was observed in three @entity1 , loss of functional focal activation in six @entity1 and @entity10 during oligemia in four @entity1 . The oligemia was observed in all seven @entity1 . It started at the posterior pole of the brain and gradually spread anteriorly in the course of 15-45 minutes. The findings indicate that a simple vasospastic model of the classical @entity2704 attack is not likely. In six @entity1 with common @entity2704 , attacks were induced with red wine. On eight occasions rCBF was measured before and after provocation, and just before and during induction of @entity2704 attacks. In three @entity1 stationary detection and @entity4185 was used. In one @entity1 intra-arterial xenon and in four @entity1 @entity4185 and 133-xenon tomography measuring from about 700 regions of the brain. Measurements were taken about 15 minutes apart. In no @entity1 was oligemia observed at any time. The results suggest that the pathogenesis of common @entity2704 is different from that of classical @entity2704 .
