Chronic XXXX as a possible sequel to encephalitis.

@entity1 @entity2086 @entity1032 @entity304 @entity294 @entity8442 @entity16 @entity1394 @entity302 @entity11173
A @entity1 is reported who presented with a typical @entity8442 . Before this illness, the @entity1 had suffered from @entity11173 at the age of 15 months. A @entity1394 was present which included severe @entity1032 , @entity304 , and @entity302 . There were no relatives with a similar disease. Clinically, there was a frank recurrent @entity8442 with occasional @entity294 . Radiographic barium studies showed delayed transit, hypomotility of the intestines, and gross distension of the esophagus, stomach, small intestine, and colon. Histological examination of the gastrointestinal tract revealed a normal appearance of the mucosa; however, there was a @entity2086 with abnormalities of the plexuses. Both the submucosal and the myenteric plexuses were reduced in number and size. They showed a decreased number of ganglion cells, proliferation of Schwann cells and infiltration by lymphocytes. The abnormalities were most strikingly present in the esophagus and the small intestine. The @entity16 resulting in clinical pseudoobstruction is proposed to be part of the generalised neurological pathology as a late sequel to the @entity11173 .
