Reproductive risk factors for XXXX by receptor status, histology, laterality and location.

@entity1 @entity1137 @entity0 @entity5 @entity189 @entity513
It is well established that a @entity1 's reproductive history influences her risk of @entity0 . We examined whether the effect of reproductive history was similar for different sub-types of @entity0 . The study was based on a population-based cohort of 1.5 million Danish @entity1 born between 1935 and 1978, with individual information on births. Between 1978 and 1994, 10,790 incident cases of @entity0 were identified in a nationwide @entity5 registry, including detailed information on receptor status, histology, laterality and location of the @entity5 . Overall, the incidence of @entity0 was 13% lower in parous compared with nulliparous @entity1 . This reduction was significantly stronger for mucinous than for @entity513 and for @entity5 located centrally than for those non-central in the breast. Overall, the incidence in parous @entity1 increased by 10% by each 5-year postponement of their first birth. For the incidence of @entity1137 this increase was significantly stronger, and for @entity189 it tended to be stronger than for @entity513 . For the incidence of centrally located @entity5 the increase was stronger than for non-centrally located @entity5 . On average, there was a 10% decrease in @entity0 risk by each additional birth. This decrease was seen in most sub-types, but not for @entity1137 of for centrally located @entity5 . According to our findings, lobular and @entity189 and centrally located @entity5 may have risk-factor profiles that differ from other types of @entity0 .
