[Autopsy case of Henoch-Sch  nlein XXXX complicated with intestinal @entity2803 ].

@entity1 @entity644 @entity1985 @entity2803 @entity417 @entity548 @entity1333 @entity4 @entity1066 @entity4125 @entity134 @entity205 @entity7860 @entity490 @entity1011 @entity4841 @entity811 @entity7622 @entity360
A 55-year-old @entity1 was admitted to our hospital because of @entity4841 , @entity4125 , @entity490 , melena and leg @entity4 . Laboratory findings showed an increased serum @entity1011 level (2.4 mg/dL), @entity811 and massive @entity644 (10.7 g/day). Renal biopsy revealed diffuse endocapillary proliferation and focal @entity7622 with IgA deposition predominantly in the glomerular capillary walls. Based on these findings, he was diagnosed as having Henoch-Sch  nlein @entity7860 and @entity134 therapy was started. Following @entity134 therapy, his @entity1985 state remained unchanged, although his renal function improved concomitantly with the disappearance of @entity4841 , @entity4125 and abdominal symptoms. Therefore, @entity205 was added to the @entity134 therapy to enhance immunosuppression. However, melena recurred and @entity417 progressed. Endoscopy revealed @entity1066 in the duodenum and jejunum, and clipping was performed at some @entity548 sites. However, he died of @entity548 . The autopsy revealed that @entity548 having @entity2803 spread widely in the stomach, duodenum and jejunum. Recurrence of @entity360 during the treatment of Henoch-Sch  nlein @entity7860 is usually due to severe @entity1333 or @entity134 ulcer. However, in @entity1 receiving strong immunosuppressive therapy, @entity2803 needs to be considered as cause of @entity360 .
