Does XXXX contribute to hemifacial spasm?

@entity1 @entity407 @entity101 @entity6 @entity2850 @entity731 @entity62 @entity4288 @entity1649
BACKGROUND: @entity1649 ( @entity1649 ) is caused by @entity2850 of the root exit zone (REZ) of the facial nerve. However, the mechanism that causes the offending vessels to compress the REZ has not been clarified. Elongation of intracranial arteries due to @entity4288 is one possibility, but such arteriosclerotic changes are not observed very frequently among @entity1 with @entity1649 . The aim of the present study was to investigate whether arteriosclerotic changes would contribute to the pathogenesis of @entity1649 . METHODS: This study included 111 @entity1649 @entity1 , all of whom were Japanese. The prevalence rates of @entity101 , @entity407 , and @entity6 were examined as risk factors of @entity731 , and the cardio-ankle vascular index (CAVI) was measured as an indicator of arteriosclerotic change. The severity of @entity62 ( @entity62 ) in @entity1649 @entity1 was measured by magnetic resonance imaging. These data were compared with data from healthy Japanese controls. RESULTS: The prevalence rates of the risk factors for @entity731 in the @entity1649 @entity1 were not higher than those in the general Japanese population. The CAVI scores for the @entity1649 @entity1 were similar to, or lower than those in the healthy controls for all age groups except 60 to 69-year-old @entity1 . The severity of @entity62 in the @entity1649 @entity1 was not significantly worse than that in the controls. CONCLUSIONS: It is suggested that arteriosclerotic changes are not involved in the pathogenesis of @entity1649 , and that @entity2850 are attributable to anatomical features of the intracranial arteries and facial nerves formed during the prenatal stage.
