Sleep and XXXX .

@entity1 @entity410 @entity1949 @entity12924 @entity73 @entity739 @entity4939 @entity1654 @entity2473 @entity302 @entity751 @entity3148 @entity4940 @entity159
UNASSIGNED: Sleep and @entity302 have a close relationship. About 20% of @entity1 suffer @entity410 only during the night, approximately 40% only during the day and approximately 35% during the day and night. In @entity302 , the occurrence of @entity410 is strongly related to sleep or awakening. Infantile @entity2473 appear predominately on awakening, and @entity739 is sometimes visible only in sleep. @entity1 with @entity751 or @entity302 with @entity1654 (BECTS) have @entity410 mostly when asleep, and in both syndromes interictal spike waves are markedly accentuated in slow wave sleep. Electrical @entity159 during slow sleep/continuous spike wave discharges during sleep (ESES/CSWS), atypical benign partial @entity302 , and @entity12924 are @entity302 with substantial behavioral and @entity73 , @entity410 , and continuous spike-wave activity during non-rapid eye movement (NREM) sleep. The hallmark of juvenile @entity3148 and grand mal @entity410 on awakening are @entity410 symptoms within 2 hours after awakening, often provoked by @entity1949 . @entity4940 is sometimes mistaken for @entity4939 . Differentiation is possible when the clinical symptoms and the frequency of the paroxysmal events per night and month are carefully observed and nocturnal video electroencephalography (EEG) performed. Sleep EEG recordings may be helpful in @entity1 with suspected @entity302 and nonconclusive awake EEG. Depending on the clinical question, sleep recordings should be performed during nap (natural sleep or drug induced), during the night, or after @entity1949 .
