Late XXXX 16 days after delivery: case report with clinical, radiologic, and pathophysiologic correlations.

@entity1 @entity644 @entity410 @entity101 @entity2399 @entity4 @entity409 @entity3036 @entity5052 @entity892 @entity2553 @entity10 @entity427
BACKGROUND: @entity3036 is a rare, frightening, and potentially tragic complication of @entity101 pregnancies, usually developing within 48 hours of delivery. @entity410 occurring days to weeks after parturition are exceedingly uncommon and require rapid, precise clinical evaluation by multiple specialists. METHODS: A case presentation of delayed @entity3036 illustrates unique features of the syndrome. Extensive review of the literature highlights pathogenesis, controversies, and dilemmas surrounding this enigmatic @entity101 . RESULTS AND CONCLUSIONS: A 39-year-old @entity101 @entity1 had an uneventful full-term delivery by her family physician only to develop @entity10 , @entity892 , and recurrent @entity410 16 days later. Initial evaluation showed severe @entity101 , @entity892 , @entity427 , @entity644 , and @entity5052 . She was given a @entity2399 infusion. Magnetic resonance imaging (MRI) documented asymmetric ischemic foci within gray matter in the distribution of the posterior cerebral arteries. All symptoms, signs, and abnormal laboratory values resolved within 4 days. A follow-up MRI showed complete resolution of all cytotoxic cortical lesions. Based on @entity1 autopsy data, radiologic investigations, and animal studies, @entity3036 is believed to result from explosive @entity2553 , @entity409 , and cytotoxic @entity4 of cerebral cortex. This central nervous system vasculopathy is most prominent in the posterior cerebral vasculature and is often rapidly reversible. Difficulties in differential diagnosis, typical findings on neuroimaging, and urgent management strategies are discussed. The time limit for @entity3036 probably should be lengthened to 4 weeks, as indicated by our case and other clinical series.
