[ XXXX following continuous ambulatory peritoneal dialysis].

@entity1 @entity2976 @entity5217 @entity11243 @entity1919 @entity413 @entity298 @entity1928
@entity2976 is a severe complication after CAPD treatment. The visceral peritoneum is thickened and interenteric adhesive parts are found. Myofibroblasts are proliferated and the collageneous tissue is hyperplastic. The mean clinical symptom is the mechanical obstruction of the small bowel. We observed this illness in three out of sixty @entity1 under CAPD. These @entity1 had higher incidence of @entity298 . In the ascites high concentrations of PG E2 and Thromboxan B2 were observed. After treatment of the infection the concentrations fell down to normal values. Electronoptical observations from peritoneal biopsies showed a proliferation of myofibroblasts and extracellular lysosomes. It is known from these lysosomes that they are able to set free proteasis. These lead to degredation of fibrin and fibrinogen. These splits are mitogen to myofibroblasts. release from HIT cells could also be evoked by the sulphonylureas @entity5217 and @entity11243 and by an increase in concentration of extracellular K+ to 40 mmol/l. The content of @entity1919 in HIT cells was increased modestly by @entity413 but not by an increase in extracellular K+. @entity1928 elicited a 4-fold increase in @entity1919 content. We conclude that HIT cells retain the essential features of the insulin secretory response of normal B cells and represent an important tool for further biochemical characterisation of the secretory system.
