[Functional morphology of XXXX ].

@entity1 @entity712 @entity296 @entity301 @entity95 @entity519 @entity453 @entity299 @entity731
OBJECTIVE: To show that @entity296 ( @entity296 ) that is generally believed to be a variant of @entity299 ( @entity299 ) and an analogue of chronic @entity712 is an initial stage and the background of all further forms of @entity299 . SUBJECTS AND METHODS: Sixty cardiac biopsies obtained from @entity1 during aortocoronary bypass surgery for @entity299 and myocardia from 55 @entity95 in which the investigators simulated @entity731 over time, as well as acute, transient, and recurrent @entity301 after vasopressin administration were examined. A number of adequate histological, histochemical, and immunohistological procedures, electron microscopic and electron histochemical studies, and morphometry were employed. The investigators made biochemical monitoring of blood cholesterol and triglyceride levels and ECG monitoring in the animals had received intravenous vasopressin. RESULTS: The authors have indicated that @entity299 begins with endothelial changes in the trophic component of the microhemocirculatory bed, which gives rise to its impaired blood flow and to @entity519 at the level of the myocardial structural and functional unit--fascicules. Over time, the disease develops a set of processes that cause damage to microvessels, cardiomyocytes, stroma, lymph capillaries, and nerve endings and changes in secretion. Compensatory and adaptive responses that long mask the symptoms of damage to the vascular bed and myocardial tissue take place simultaneously. The occurring lesions result in irregular diffuse cardiosclerosis, micro- and lymphocirculatory, innervation, and conduction disturbances, and irregular cardiomyocyte damage. The authors consider this set of myocardial changes to be an initial phase of @entity296 that shows no clinical manifestation at all. As @entity453 progresses, the already damaged myocardium is superimposed by the morphological changes caused by higher @entity519 at the whole organ level. Depending upon a combination of these changes, one or another form of @entity299 and progression of chronic @entity299 may prevail at a later time. CONCLUSION: @entity296 is a broader concept than chronic @entity712 and it is a basis for the pathogenesis and morphogenesis of @entity299 .
