Delayed XXXX following @entity510 : Can the role of severe angiographic @entity2553 really be dismissed?

@entity1 @entity2806 @entity510 @entity63 @entity1188 @entity2553
BACKGROUND: The recent literature pertaining to delayed @entity2806 following @entity510 has downplayed the role of angiographic @entity2553 . However, it is our hypothesis that angiographic @entity2553 has a significant pathophysiological role in this disease. We undertook an observational radiographic study of @entity1 who presented in a delayed manner (>72   h from ictus) with evidence of severe angiographic @entity2553 on initial angiography in order to describe an apparent association between @entity2553 and @entity1188 location. METHODS: This was a retrospective study of consecutive @entity1 treated at our unit. Initial, subsequent, and follow-up cross-sectional imaging with CT or MRI was analyzed in conjunction with initial angiography. Sites of angiographic narrowing, angiographic hypoperfusion, and subsequent sites of @entity1188 were assessed. RESULTS: Thirteen @entity1 (6 @entity1 , 7 @entity1 ) of mean age 49   years were assessed. Mean time to presentation was 6   days. All had severe angiographic @entity2553 . Nine of the 13 @entity1 suffered @entity1188 ; the @entity1188 in seven of the nine @entity1 were large. There was correlation between sites of angiographic narrowing and @entity1188 in all cases and eight of the nine cases showed angiographic hypoperfusion in a location corresponding to eventual @entity1188 location. CONCLUSIONS: Severe angiographic @entity2553 may be linked to @entity1188 in @entity1 who present late. These @entity1188 are mostly large despite maximal treatment. We question the notion that proximal @entity2553 has a minor role in delayed @entity63 .
