Oliguric @entity810 in XXXX with @entity2617 in the kidneys.

@entity1 @entity4888 @entity8 @entity644 @entity4658 @entity101 @entity2617 @entity1458 @entity177 @entity810 @entity1986 @entity811 @entity551
OBJECTIVE: To present the clinical picture of @entity810 in @entity1 with @entity4888 ( @entity4888 ) and @entity8 . To analyze the pathogenesis of @entity177 . METHODS: Correlation of clinical picture, urinary findings, imaging reports and autopsy findings in two @entity1 with long-standing @entity4888 who died with @entity177 . CASE SUMMARIES: Both subjects had sustained @entity1458 in the last 2 weeks. One @entity1 had persistent @entity551 , normal urinalysis, normal renal sonogram, and scarce @entity1986 with preservation of renal parenchymal architecture. He was thought to have ischemic @entity810 not directly linked to the @entity2617 . The second @entity1 developed @entity101 one month prior to death, and had moderate @entity644 , @entity811 , @entity4658 , grossly enlarged kidneys with hypoechoic masses, and extensive replacement of the renal parenchyma by @entity2617 . This picture is typical of @entity177 secondary to lymphomatous replacement of the kidneys. CONCLUSIONS: The development of @entity177 in @entity4888 with @entity8 may have varying clinical and imaging manifestations and pathogeneses. Potentially reversible pathogenic mechanisms should be systematically investigated, particularly if the overall clinical picture is not characteristic of @entity177 secondary to lymphomatous replacement of the parenchyma.
