The influence of gender on XXXX .

@entity1 @entity1164 @entity4710 @entity1166 @entity5126 @entity955 @entity453 @entity943
It is becoming increasingly apparent that there are important gender differences in normal cardiac physiology. These, in turn, could be associated with differences in the type and severity of @entity943 . @entity1 have higher resting heart rates than @entity1 , probably as a result of a combination of autonomic and intrinsic factors. The clinical significance of this observation is unclear at the present time. @entity1 have a lower incidence of @entity1164 , which could be related to the delayed onset of @entity453 in @entity1 , presumably as a result of the protective effects of female hormones during gestational years. In survivors of @entity1164 , there are major gender differences, with fewer @entity1 having underlying @entity453 and a greater percentage of @entity1 having structurally normal hearts. QT interval prolongation and @entity1166 are more common in @entity1 , probably on the basis of differences in ion channels between genders. @entity1 appear especially susceptible to Torsades from QT-prolonging drugs such as @entity5126 or tricyclic antidepressant medications. The mechanisms of paroxysmal @entity4710 differ between the genders, although therapy seems to be equally efficacious in @entity1 and @entity1 . Lastly, @entity955 is considerably more common in @entity1 . There is also some evidence that it is better tolerated by @entity1 .
