XXXX as cause for focal @entity51 .

@entity1 @entity410 @entity717 @entity51 @entity911 @entity302 @entity1662 @entity159
In a four-and-a-half year period, 19 @entity1 with focal @entity51 accompanied by a focal @entity159 were encountered. Sixteen of these @entity1 showed clouding of @entity1662 or @entity911 . Computed tomography of the brain revealed focal lesions in 15 @entity1 . In 7 @entity1 the lesions were the result of a recent cerebral event and in 8 @entity1 they were long-standing. All @entity1 were treated with anti- @entity302 drugs. Ten of the 12 @entity1 without an acute lesion showed a complete recovery in a few days. In these @entity1 the symptoms may have been caused by the continuous @entity410 activity, classifying them as cases of non- @entity410 focal @entity159 . Only 2 of the 7 @entity1 with an acute lesion had a full recovery. In @entity1 with an acute lesion the part played by the @entity159 in the acquired deficits is unclear. Continuous or frequent intermittent focal @entity302 discharges on the EEG may warrant treatment with anti- @entity302 drugs in @entity1 with focal @entity51 , even when one of the hallmarks of @entity302 , @entity717 , is absent.
