@entity1276 in XXXX . Pathogenic and therapeutic considerations.

@entity1 @entity6583 @entity1276 @entity2318 @entity693 @entity302
The cases of 11 @entity1 with @entity1276 secondary to @entity6583 are analyzed. Most of the @entity1 had suffered from @entity302 before they developed hydrocephalic symptoms, and computerized tomography showed that infestation of the parenchyma coexisted with ventricular or cisternal colonization. In four cases, the parasitic vesicles compromised cerebrospinal fluid (CSF) flow in the ventricular system, resulting in internal @entity1276 . Communicating @entity1276 , caused by the presence of Cysticercus larvae in the basal cisterns (Cysticercus racemosus), or by the occurrence of a @entity693 , or both, developed in seven more @entity1 . Changes in CSF pressure were related to the number and location of the cysts and to the leptomeningeal inflammatory reactions evoked by them. The majority of @entity1 presented with a chronic and relatively normotensive @entity1276 . All @entity1 except one had identifiable ventricular or cisternal Cysticercus larvae; these @entity1 were treated with open removal of the cysts, and did well. However, most of them had impairment of CSF flow through the basal cisterns and required permanent CSF shunting. Communicating @entity1276 due to @entity2318 was also successfully managed with extracranial shunting. @entity302 was controlled with anticonvulsant therapy. Although good lasting results may be obtained with aggressive treatment of @entity6583 , @entity1 are liable to relapse because surgery is only palliative in most instances.
