XXXX . A review.

@entity129 @entity455 @entity6 @entity73 @entity4401 @entity262 @entity4091
@entity455 involving @entity129 are reviewed in this study. These are indicated by the correspondence between groupings of clinical manifestations and anatomical vascular distributions. Confirmatory evidence is provided by mishaps, following diagnostic or therapeutic angiography. Three systems play a role in the vascularization of cranial nerves: the inferolateral trunk (ILT), most often arising from the internal carotid artery, the middle meningeal system (MMS), and the @entity262 ( @entity262 ); the latter two are both derived from the external carotid artery. Conclusions concerning the ILT are least definite because of variations in vascular territory and the lack of confirmation from embolic events in a vascular region that is rarely the site of embolization. The specific ILT territory includes @entity129 , VI and V1. Knowledge of the vascularization of nerve III may furnish explanations as to its different modes of @entity6 . For the MMS, the cranial nerves concerned are V2, V3 and VII. Two vascular territories for the intrapetrous portion of nerve VII are defined: the first (stylomastoid artery) is limited to nerve VII; the second (MMS) supplies VII and V. Two examples of involvement of VII and V following selective MMS embolization are presented. A study of @entity4401 associated with nerve @entity73 is summarized. The @entity262 supplies nerves IX, X, XI and XII; XI has a dual vascularization which explains why it can either be spared (as was the case in an angiographic accident) or involved (as in a case of @entity4091 ). A vascular mechanism should be considered when @entity129 occur in the syndromes described here.
