Morphological changes associated with hemodynamically significant myocardial bridges in XXXX .

@entity1164 @entity296 @entity82 @entity301 @entity4 @entity509 @entity731
Although @entity296 ( @entity296 ) is a common @entity301 , its cardiovascular consequences are still disputed. A positive link between @entity1164 ( @entity1164 ) and @entity296 has not yet been definitively proved, even though many case reports and small scale studies support this association. For @entity296 to be associated with @entity1164 it must exhibit certain specific characteristics involving coronary or myocardial changes sufficient to explain a terminal cardiac event. In this study we aimed to analyze the morphological changes (both myocardial and coronary) associated with hemodynamically significant @entity296 and the morphological differences between hemodynamically significant @entity296 and @entity296 considered to be non-hemodynamically significant. MATERIAL AND METHOD: We analyzed 53 cases of @entity1164 , of which 21 cases had hemodynamically significant @entity296 , 14 had non-hemodynamically significant @entity296 and 20 cases suffered @entity1164 without @entity296 , using a morphological score with seven histological parameters. RESULTS: @entity296 and @entity4 were found to be highly correlated with hemodynamically significant @entity296 (HSMB), as were @entity4 and @entity82 . CONCLUSIONS: Hemodynamically significant @entity296 can be discovered during heart dissection by analyzing a series of morphological markers (width, distribution of @entity731 , @entity509 ). Our study showed that @entity296 was associated with @entity296 and @entity4 , both of which have an increased risk of electrical instability. Compared to non-hemodynamically significant @entity296 , HSMB shows a distinct histological pattern, with @entity296 and @entity4 . The main cause of @entity1164 in association with HSMB seems to be electrical due to increased electrical myocardial heterogeneity, but large scale studies are needed to test this.
