Changing trends in the etiologic diagnosis of XXXX .

@entity1 @entity64 @entity887 @entity1031 @entity385
The evaluation of victims of @entity385 has evolved over the last few years, primarily as a result of the introduction of innovative, sensitive, and informative diagnostic procedures. The role of the neurologist appears to have been redefined as one in which the identification of the presumptive cause and mechanism of the @entity64 is one of the primary responsibilities. We compared the etiologic @entity64 subtypes of @entity1 entered into the Saint Louis University Stroke Registry for the period January to June, 1986 (Group A), with analogous data collected in @entity1 entered from January to June, 1992 (Group B). Group A included 57 @entity1 whose @entity64 subtype distribution was as follows: 19% due to athero- @entity1031 , 18% cardiogenic, 5% lacunar, and 4% due to other causes. In addition, 54% of @entity1 had @entity64 of unknown etiology. Group B comprised 137 @entity1 with the following etiologic distribution: 35% cardiogenic, 26% atherothrombolic, 3% lacunar, and 6% from other causes. Only 29% had @entity64 of unknown etiology. The apparent increase in the proportion of @entity64 may be related to wider utilization of transesophageal echocardiography. There was also a significant drop in the proportion of @entity64 of unknown etiology. Finally, in comparison with other @entity64 registries, our data suggest that many small @entity64 may also result from @entity887 . These apparent changes in the proportion of @entity64 subtypes should be considered when @entity1 are being evaluated for risk stratification.
