Evaluation of @entity296 through serum troponin I and echocardiography in XXXX .

@entity1 @entity296 @entity341 @entity9576 @entity661 @entity751 @entity299 @entity2021
BACKGROUND: Anaphylaxis is an acute, @entity9576 that results from the sudden release of mast cell- and basophil-derived mediators. Although @entity2021 can cause @entity299 , the incidence of @entity296 using troponin I (TnI) has not been characterized. In addition, patterns of @entity296 have not been evaluated in @entity1 with elevated TnI. Therefore, we studied the occurrence and patterns of @entity296 with TnI and echocardiography in @entity2021 . METHODS: We conducted a retrospective review of 300 consecutive @entity2021 cases that were diagnosed in the @entity341 ( @entity341 ) over 53 months (2011-2015). @entity296 was defined as elevation of TnI within 24hrs after arrival at the @entity341 . We investigated systolic function and regional wall motion abnormality (RWMA) through echocardiography within 5 hours after arrival at the @entity341 in @entity1 with @entity296 . RESULTS: Among 300 @entity1 (median age, 55 years), 22 @entity1 demonstrated @entity296 (7.3%). The median TnI was 0.222 ng/mL with a range from 0.057 ng/mL to 19.4 ng/mL. Three @entity1 presented reduced systolic function and 4 @entity1 showed RWMA. One @entity1 showed reverse @entity661 and other 2 @entity1 had RWMA discordant to the distribution of coronary arteries. Another @entity1 showed RWMA (inferior wall) with ST elevation of II, III, and aVF. All 4 @entity1 were discharge after recovery of @entity296 without any specific intervention for @entity296 . CONCLUSIONS: @entity296 developed in 7.3% of @entity1 with @entity2021 . Various @entity296 , including @entity751 and @entity661 , has been observed in @entity1 with @entity296 .
