Diet and XXXX and rectum: a case-control study.

@entity1 @entity167 @entity5 @entity620 @entity904 @entity6741 @entity14 @entity105 @entity694
In 1979-81, 419 @entity1 with incident cases of @entity694 and 732 controls were questioned regarding diet and @entity167 . @entity5 cases were a population-based series reported to the South Australian Central @entity5 Registry, were 30-74 years of age, and were residing in Metropolitan Adelaide. Controls were selected from the electoral roll and individually age- and sex-matched to @entity5 cases. The most consistent risk factor for @entity14 was dietary protein, which was associated with a twofold-to-threefold relative risk for @entity694 and for @entity5 in @entity1 for all levels of consumption above the base line (i.e., the lowest consumption quintile). For male @entity694 the corresponding relative risk was similar; but for male @entity5 , risk was elevated only at old ages. Total energy intake and, less clearly, meal frequency were also positively associated with increased risk. Total @entity167 intake (but not specifically beer) was associated with increased risk of both @entity694 in @entity1 ; in both sexes, there was an increased risk of @entity694 associated with spirits consumption. A reduced risk of @entity5 was associated with @entity620 but not with @entity904 . The increased risk associated with high protein and total energy was confined to those consuming a low fiber diet, particularly among @entity1 ; but some other aspects of the relationship between fiber consumption and risk of @entity14 were more complex. Some modifications and extensions of the current fat-to- @entity6741 -to-fiber theory of bowel @entity105 were suggested.
