[Visual field defects in XXXX ].

@entity1 @entity227 @entity308 @entity1276 @entity428 @entity1805
Eight @entity1 representing @entity227 associated with @entity1276 are reviewed. Seven cases had @entity1276 and one had congenital communicating @entity1276 . We found five cases of defects in @entity227 : (1) inferior @entity1805 with inferior nasal quadrantanopia in the opposite eye; (2) inferior binasal quadrantanopia; (3) @entity308 ; (4) unilateral temporal defect; (5) bilateral central scotoma. In these cases CT demonstrated moderate or marked symmetrical dilatation of the third and lateral ventricles. Four out of five cases showed bulging of the third ventricle anteriorly into the sella turcica on CT or ventriculography. Other three @entity1 had @entity1805 . Characteristic asymmetrical dilatation of the lateral ventricles was noted in all three cases. The more enlarged lateral ventricles were ipsilateral with the affected visual pathways. The sites of lesion responsible for these field defects seemed to be optic tract in one case and optic radiation in two cases. Ventriculoperitoneal shunt was placed in five out of seven cases. Impaired visual field improved in three @entity1 after shunt insertion. A 28-year-old female who had history of blurred vision fos 14 days showed improvement in visual acuity and field when the enlarged ventricles became slit-like by shunting. In the other two @entity1 defects in visual fields improved in spite of consistent @entity1276 . These facts suggested that not only the mechanical forces with distended third ventricle but also @entity428 played an important role in producing @entity227 in hydrocephalic @entity1 .
