Postoperative inflammation in the abdominal cavity increases adhesion formation in a laparoscopic XXXX model.

@entity32 @entity19 @entity516 @entity515 @entity619 @entity1104
OBJECTIVE: To investigate acute @entity32 in the peritoneal cavity in adhesion formation. DESIGN: Prospective randomized, controlled trial. SETTING: University laboratory research center. ANIMAL(S): 9- to 10-week-old BALB/c female @entity19 . INTERVENTION(S): In a laparoscopic @entity19 model, acute @entity32 in the peritoneal cavity evaluated in @entity515 @entity516 enhanced @entity1104 , by @entity515 @entity516 plus manipulation, and in the latter group plus @entity619 . MAIN OUTCOME MEASURE(S): Qualitative and quantitative adhesion scores and an acute @entity32 score (neoangiogenesis, diapedesis, and leukocyte accumulation). RESULT(S): Adhesions at the lesion site were enhanced by the @entity515 @entity516 , further enhanced by manipulation, and decreased by the administration of @entity619 . The acute @entity32 scores (total, neoangiogenesis, diapedesis, and leukocyte accumulation) strongly correlated with the total adhesion score. @entity32 scores were similar at both the surgical lesion and the parietal peritoneum. CONCLUSION(S): Acute @entity32 of the entire peritoneum cavity is an important mechanism involved in adhesion formation and enhances adhesion formation at the lesion site.
