@entity9927 : an ultrastructural study of reaction to injury in the XXXX alveolar wall.

@entity1 @entity1223 @entity3126 @entity4530 @entity3265 @entity4 @entity384 @entity32428 @entity651 @entity9927 @entity891 @entity3789 @entity505 @entity130
This study was undertaken to determine the site of initial @entity651 in @entity9927 . A hotel fire in Houston, Texas, resulted in the on-site deaths of 10 white @entity1 (2 to 62 years of age). All underwent autopsy examinations which included measurement of @entity1223 (CO) and @entity3789 (CN) levels, as well as electron microscopy of lung samples. Average CO levels of 40% and CN levels of 0.6 ppm were obtained. In all cases, the lungs were heavy, hyperemic, and @entity4 with soot staining the tracheobronchial mucosa. Light microscopy showed soot, @entity891 , and @entity4 . Electron microscopy confirmed the presence of interstitial and @entity384 and @entity4 . @entity505 particles were also present, and occasionally were seen undergoing phagocytosis by alveolar macrophages. Intracellular @entity4 with @entity4530 and vesicle formation was prominent within Type I pneumocytes in 9 of 10 cases. Endothelial cells showed similar but much less severe changes, lacking the distinct blebs seen in the Type I cells. This investigation reveals that @entity9927 , like @entity3265 inhalation and @entity32428 instillation, appears to cause @entity3126 by @entity130 to the Type I pneumocyte.
