Severe @entity339 may prevent mural XXXX formation within the left ventricle with @entity299 .

@entity1 @entity339 @entity66 @entity384 @entity910 @entity439 @entity5707 @entity299 @entity715 @entity1002
The protective effect of severe @entity339 ( @entity339 ) against left @entity715 formation has been well documented. It was also proposed that severe @entity339 may prevent @entity715 formation within the left ventricle (LV) with @entity299 . Therefore, we investigated whether ischemic @entity339 prevents @entity715 formation within the LV in @entity1 with @entity299 . The study population was comprised of 1313 @entity1 (1133 males, 180 females, age 56+/-18) with @entity1002 documented by coronary angiography and left ventriculography. None of the @entity1 had a history of chronic anticoagulation. Epicardial coronary arteries were normal in 91 @entity1 , and single-vessel, two-vessel, and @entity66 were detected in 328, 330, and 564 @entity1 , respectively. @entity1002 and severe @entity339 were detected in 191 (14.5%) and 125 (9.5%) @entity1 , respectively. Overall incidence of @entity5707 was lower in @entity1 with severe @entity339 than in @entity1 without severe @entity339 (4% vs 15.6%, OR: 0.2, P<0.001). Severe @entity339 compared with absence of severe @entity339 was associated with a lower incidence of @entity5707 both in @entity1 with @entity384 (6.8% vs 34.2%, OR: 0.19, P<0.001), and in @entity1 with @entity439 (3% vs 18%, OR: 0.14, P<0.0001) involving anterolateral, septal and/or apical LV segments. A similar trend without statistical significance was also observed in @entity1 with @entity910 (4.7% vs 16%, OR: 0.26, P=0.1) related to anterolateral, septal and/or apical LV segments. However, @entity339 had no impact on the incidence of @entity5707 in @entity1 with @entity439 or @entity910 related to posterior and/or inferior segments (3.7% vs 3%, OR: 1.2, P>0.05). In conclusion, severe @entity339 seems to prevent LV mural @entity715 formation in @entity1 with @entity384 , and in @entity1 with @entity439 related to anterolateral, septal, and/or apical LV segments. This relative risk reduction may be associated with diastolic volume overloading due to severe @entity339 which may overcome stagnation and a procoagulant state within the LV with severe @entity299 .
