The postprandial portal flow is related to the severity of @entity101 and XXXX .

@entity1 @entity82 @entity101 @entity548 @entity450 @entity1575
BACKGROUND/AIMS: Diminished postprandial portal @entity450 has been demonstrated by echo-Doppler flowmetry in @entity1 with @entity1575 , but its diagnostic role is unclear. This prospective study was therefore undertaken in @entity1 with varying severity of @entity101 and degree of @entity1575 . METHODS: Portal flowmetry was performed in 66 @entity1 with @entity82 and 20 healthy volunteers during fasting and 30 min after ingestion of a standardized meal. Hemodynamic parameters were related to the degree of esophageal varices, @entity548 , portal hypertensive gastropathy and @entity1 -Pugh score. RESULTS: The postprandial portal blood velocity increment was low in @entity1 with esophageal varices of any degree (22-24%), compared to @entity1 without varices (49%, p<0.01) and healthy controls (65%, p<0.001), but was not different in @entity1 with or without @entity548 (22% vs. 20%). In contrast, the congestion index (CI; ratio of portal vein cross-sectional area and portal blood velocity) pre-/postprandial decreased in the @entity548 group only (CI pre/ CI post 1.30+/-0.23 (no @entity548 ) vs. 0.86+/-0.29 ( @entity548 ); p<0.01). Portal hypertensive gastropathy was not related to any of the portal flow parameters. The portal blood velocity increment was comparable in controls (65%) and @entity1 with @entity1 -Pugh class A @entity82 (56%), but lower in @entity1 with class B (32%) and class C @entity82 (15%, p<0.05 vs. class A). Also, there was no postprandial decrease in congestion index in @entity1 with the most severe @entity82 (p<0.01 class C vs. class A and B). CONCLUSIONS: The postprandial rise in portal flow is inversely related to the severity of @entity101 and @entity1575 , and may be a valuable parameter with respect to the risk of @entity548 .
