[Regression of XXXX in @entity101 ].

@entity1 @entity82 @entity101 @entity63 @entity1184 @entity4346 @entity299 @entity943
PATHOPHYSIOLOGY AND THERAPY: @entity4346 represents an important factor determining the prognosis of @entity101 @entity1 . @entity1184 as identified by electrocardiography (Table 1) or echocardiography (Table 2) characterizes @entity1 with a significantly increased risk of mortality and @entity943 . From the pathophysiological point of view this is based on @entity1184 of the media in resistance vessels, on @entity82 , on a reduced coronary flow reserve and on the occurrence of @entity63 (Figure 1). The diastolic and (later) systolic function of the heart are disturbed (Figures 2 to 4). Antihypertensive therapy with beta blockers and diuretics leads to a reduction of left ventricular mass by 5-8%, with ACE-inhibitors and AT-blockers by 13% (Figure 5). Particularly ACE-inhibitors can effectively reverse of the above mentioned pathological processes. Regression of @entity1184 goes along with an improved prognosis and a reduction of @entity943 (Figure 6). A symptomatic treatment of @entity943 should always be accompanied by medical therapy aimed at regression of @entity1184 . Optimal therapy results in normalizes of blood pressure, leads to a regression of @entity1184 and induces @entity299 , which in turn improve left ventricular function, reduces microvascular @entity63 stress and @entity943 . These therapeutic desiderates are also pertinent for @entity101 in the prehypertrophic state, as in juvenile @entity101 .
