Streptococcal origin of a case of XXXX .

@entity1 @entity644 @entity1310 @entity697 @entity1289 @entity1229 @entity786 @entity4341 @entity4125 @entity7622 @entity5417 @entity7860 @entity490 @entity631 @entity617
We report the case of a 25-year-old @entity1 with @entity490 , @entity4125 on the legs and @entity644 occurring 2 weeks after @entity697 , and admitted to our hospital with suspected Henoch-Sch  nlein @entity7860 ( @entity5417 ). He had increased anti-streptolysin O (ASO) titer and hypocomplementemia. A renal biopsy specimen showed endocapillary proliferative changes, which are typical of @entity631 ( @entity631 ). However, immunofluorescence study revealed predominant IgA and C3 deposits in @entity7622 , indicating a diagnosis of @entity5417 . Because of massive @entity644 initially, the treatment with a combination of @entity617 , @entity786 , @entity4341 and @entity1229 was started along with 3 plasma exchanges. @entity1289 inhibitor was also given. Response to the treatment was favorable. A follow-up biopsy was performed 8 months after the first biopsy. The renal biopsy specimen showed a figure of typical @entity5417 . To further investigate the cause of glomerular changes in our @entity1 , an immunofluorescent study of nephritogenic @entity1310 -associated plasmin receptor (NAPlr) of group A, beta-hemolytic streptococci was performed. NAPlr was significantly positive in the glomeruli in the first biopsy specimen, but not in the second. His clinical course and pathological findings suggest that NAPlr may be related to the pathogenesis in a part of @entity1 with @entity5417 , especially in @entity1 with high ASO titer and hypocomplementemia.
