Exercise-induced sustained symptomatic XXXX : incidence, clinical, angiographic and electrophysiologic characteristics.

@entity1 @entity1165 @entity583 @entity299 @entity3044 @entity453 @entity67 @entity943
Among 112 @entity1 with sustained @entity1165 , 15 were found to have exercise-induced symptomatic @entity1165 . This population was divided into two subgroups: group 1A included five @entity1 with @entity453 and group 1B consisted of 10 @entity1 with no structural @entity299 . All @entity1 underwent clinical examination, exercise electrocardiography, left ventriculography, coronary angiography (n = 14) and electrophysiologic study. In group 1B, right ventriculography (n = 7), M mode and two-dimensional echocardiography were also obtained. Group 1A @entity1 were compared with a population of 27 @entity1 with @entity453 and chronic sustained @entity1165 not related to exercise (group 2). There were no statistically significant differences between group 1A and group 2 in terms of age, sex, incidence of prior @entity583 , NYHA functional class, angina pectoris, symptoms during @entity943 , severity and extent of coronary arterial lesions, @entity67 and wall motion abnormalities. In group 1B, coronary angiography and right and left ventricular function were normal. During electrophysiologic study, @entity1165 was initiated in four group 1A @entity1 . In group 1B, @entity1165 was initiated in eight @entity1 . In four of these @entity1 ventricular pacing had to be combined with @entity3044 administration. In group 2, @entity1165 was induced in 26/27 @entity1 . From this study we conclude that in @entity1 with @entity453 the electrophysiologic substrate of exercise-related sustained @entity1165 does not differ from the substrate of non-exercise-related @entity1165 . Re-entry is the most likely electrophysiologic mechanism. In @entity1 without structural @entity299 , the mechanism of the @entity943 remains speculative.
