[Regional cerebral blood flow in @entity35 with XXXX produced by bilateral vertebral and carotid artery occlusion].

@entity35 @entity2806 @entity13279
Regional cerebral blood flow ( @entity13279 ) was measured prior to, during and after global @entity2806 in @entity35 . @entity2806 was produced by 10 or 30-min occlusion of both common carotid arteries which was done 24 hr after the permanent electrocauterization of bilateral vertebral arteries. The @entity13279 was measured using the radioactive microsphere technique. In @entity35 subjected to 10-min @entity2806 , @entity13279 in 9 brain regions was reduced to 11.3-54.8% (mean: 26.9%) of that of the sham operated control. Ten min after recirculation, hyperperfusion was observed in the cerebral cortex, hippocampus and striatum, and a moderate recovery was detected in the n.accumb. + olfactory tub., thalamus and hypothalamus. However, @entity13279 in these 6 regions was again decreased 20-30 min later, and it recovered to levels more than 50% of the control 60 min after the ischemic event. In the other 3 regions (cerebellum, colliculus sup. + inf., pons + medulla), @entity13279 increased toward the control level gradually, and it completely recovered 60 min after recirculation. On the other hand, in @entity35 subjected to 30-min @entity2806 , @entity13279 in 9 brain regions was reduced to 1.77-26.3% (mean: 9.6%) of that of the control. The post-ischemic hyperperfusion in the cerebral cortex and hippocampus and a moderate recovery of @entity13279 in the striatum and n.accumb. + olfactory tub. were observed 10 min after the @entity2806 . However, @entity13279 in these 4 regions remained under the control levels from 20 to 60 min after recirculation.(ABSTRACT TRUNCATED AT 250 WORDS)
