Disturbances in the XXXX metabolism in intestinal @entity63 shock.

@entity129 @entity2125 @entity63 @entity2994 @entity1425 @entity948 @entity4053 @entity432 @entity35 @entity12180 @entity4854 @entity413
Intestinal @entity63 shock is obtained in fasted @entity35 by 40-minute @entity432 ( @entity432 ) or by 35-minute @entity2994 ( @entity2994 ). Survival is prolonged by plasma treatment; further prolongation is obtained by additional administration of @entity413 . After @entity432 early @entity4854 is marked. Plasma adrenaline rises steeply after opening of the arteries and remains high, while plasma insulin remains unaltered. The @entity4854 is abolished by adrenalectomy and section of the major @entity129 (MSN) proximal to the adrenals but not by section of the MSN distal from the adrenals or by vagotomy. It is concluded that the sympathetic nervous system is stimulated by a substance, possibly related to @entity12180 , released from the intestines. After @entity2994 @entity4854 is less marked. Plasma @entity1425 as well as insulin are increased. During late and fatal @entity4053 after @entity2994 plus plasma treatment, the liver still appears to be functionally intact. It is assumed that gluconeogenesis is reversibly inhibited by as yet unknown factors. The @entity4053 cannot be abolished by injection of common substrates of gluconeogenesis but the combination @entity948 plus glucagon plus @entity2125 is highly effective.
