Posterior communicating XXXX rupture mimicking apoplexy.

@entity1018 @entity1 @entity227 @entity548 @entity1756 @entity5 @entity16 @entity439 @entity1781 @entity1410 @entity10 @entity1462 @entity438
BACKGROUND: @entity1756 can lead to devastating @entity16 and present a complex problem to treat. We report a unique case of a @entity438 (PCoA) @entity439 presenting with @entity1018 . CASE DESCRIPTION: A 39-year-old @entity1 presented with the acute onset of severe @entity10 and @entity227 loss. The @entity1 described @entity10 for several months prior to presentation. However, prior to the day of presentation, he had no visual disturbance. A CT angiogram (CTA) and magnetic resonance imaging (MRI) of the brain revealed a 1.6-cm, non-contrast enhancing suprasellar mass, eccentric to the left side, consistent with hemorrhagic mass. There was no obvious @entity439 or @entity1462 . The sella tursica was normal in appearance. The @entity1 was taken for an immediate endoscopic endonasal transtuberculum approach for @entity1781 . @entity1410 without an associated @entity5 was encountered and partially evacuated before aborting with resultant partial improvement in vision. A subsequent cerebral angiogram revealed an irregularly shaped, postero-laterally pointing, 2.5-mm left PCoA @entity439 . The @entity1 was then taken for open clipping of the @entity1756 . A large, fibrinous capsule was found over the superolateral aspect of the @entity439 . The @entity1756 was secured with clips and the surrounding @entity1410 was evacuated. CONCLUSION: In the immediate postoperative period, the @entity1 regained vision in the nasal field of his right eye. This case illustrates a unique presentation of a ruptured PCoA @entity439 , and thus must be considered in the differential diagnosis of @entity548 resulting in @entity227 in absence of a recognizable associated @entity5 .
