Vascular non-endothelial @entity842 induced by swimming exercise stress in XXXX .

@entity1808 @entity2918 @entity7293 @entity2647 @entity842 @entity35 @entity3352
1. Herein, we report the effects of acute or chronic forced swimming on vascular responsiveness to angiotensin (Ang) II. 2. The possible involvement of locally produced substances, such as @entity842 (NO) and @entity2918 , in these effects were studied in @entity35 thoracic aorta and superior mesenteric arteries. 3. Chronic, but not acute, swimming reduced the efficacy (maximal effect; Emax) of @entity1808 in thoracic aorta and mesenteric arteries, either with intact or denuded endothelium. 4. The efficacy of @entity1808 was reduced in the presence of @entity3352 in mesenteric arteries, but not in the aorta, from either control or chronically stressed @entity35 . 5. Treatment with @entity7293 reversed the effect of chronic stress on the response to @entity1808 , suggesting that chronic stress may increase non-endothelial NO activity in both the aorta and mesenteric arteries. 6. The effects of acute and chronic stress on vascular reactivity were selective for @entity1808 because no changes were observed on the effects of @entity2647 .
