Somatosensory evoked potentials in the detection of @entity2759 in XXXX repair.

@entity1 @entity3325 @entity1508 @entity1498 @entity2759
Cortical @entity3325 ( @entity3325 ) monitoring was used in 15 @entity1 2 to 50 years old undergoing repair of @entity1498 to detect the onset of @entity2759 during the cross-clamp period. Three different response patterns were observed. In 8 @entity1 (53%), the @entity3325 remained unchanged throughout the cross-clamping. This was designated a type 1 response. Six @entity1 (40%) showed a gradual deterioration in the @entity3325 after 15 minutes of cross-clamping (type 2 response). All SEPs returned to normal levels within 5 minutes of release of the clamp. One @entity1 (7%) demonstrated a decline in @entity3325 commencing prior to the application of the cross-clamp when an intercostal vessel was controlled with slings. The @entity3325 completely disappeared within 5 minutes of cross-clamping, but after 19 minutes the repair was completed and the @entity3325 returned within 3 minutes of reperfusion (type 3 response). No @entity1 sustained neurological sequelae of repair. We believe that @entity3325 monitoring offers the potential to identify the @entity1 at risk of developing @entity2759 intraoperatively before irreversible damage occurs. However, it is susceptible to deep @entity1508 anesthesia, which abolishes all cortical responses and requires expert monitoring.
