XXXX of the knee after sensory nerve injury.

@entity1 @entity128 @entity6865
@entity6865 ( @entity6865 ) of the knee is an extremely difficult problem to treat. This study examined the possible relationship between isolated @entity128 (IPBSN) and the etiology and natural course of @entity6865 . Thirty-five @entity1 with clinically significant sympathetic dystrophy of the knee were examined retrospectively. All @entity1 (100%) had clinical evidence of insult to the IPBSN. Thirty-three @entity1 (94%) were found to have vasomotor instability as measured by isolated cold stress testing (ICST). All @entity1 in this population of 33 were treated with vasoactive therapies. Subjective improvement was noted in 20 @entity1 (p = NS). Initial ICSTs of improved and unimproved @entity1 were compared. Baseline temperatures were significantly warmer in @entity1 who improved with therapy (p less than 0.05), and a warmer trend was evident throughout all phases of the test in those who improved compared with those who did not. Eighty percent of @entity1 treated within 1 year improved with one or more vasoactive therapies, whereas only 44 percent improved when treatment was started after 1 year, indicating a significant population difference (p less than 0.05).
