@entity854 in white matter, regulatory T lymphocytes, and XXXX after @entity64 .

@entity1 @entity2122 @entity64 @entity308 @entity75 @entity854 @entity385
BACKGROUND: The purpose of the present study was to investigate the existence of @entity854 in the white matter circuit in @entity64 @entity1 and its relationship to @entity308 episodes. To target the prevention of @entity308 , we also investigated the relationship between lymphocyte subsets and @entity75 in @entity1 . METHODS: @entity1 included 18 @entity1 with acute @entity385 and 22 healthy control subjects. Diffusion tensor imaging was performed. Whole-brain voxel-based analysis was used to compare fractional anisotropy (FA) between groups. Blood samples were obtained, and the lymphocyte subsets were evaluated using flow cytometry. Follow-up examinations were conducted on 12 @entity1 at 6 months. RESULTS: FA was decreased in the bilateral anterior limb of the internal capsule in @entity64 @entity1 . At the 6-month follow-up examination, there was a significant increase in FA, which was associated with a lower @entity308 scale score. @entity1 showed a decreased percentage of circulated regulatory T lymphocytes, and the degree of reduction was related to the decrease in the FA value in the internal capsule. CONCLUSIONS: FA reductions in the anterior limb of the internal capsule cause @entity2122 and confer a biological vulnerability, which in combination with environmental stressors results in the onset of @entity308 . Our findings also demonstrated the possibility of preventing @entity64 by targeting the role of regulatory T lymphocytes in brain tissue repair and regeneration after @entity64 .
