Induced @entity101 for the treatment of @entity2806 after XXXX . Direct effect on cerebral blood flow.

@entity1 @entity101 @entity2806 @entity510 @entity772 @entity439 @entity2647 @entity2553
The best treatment for symptomatic @entity2806 from presumed @entity2553 after @entity510 remains a matter of controversy. A direct effect of any treatment modality on regional cerebral blood flow has never been documented. In a series of 43 @entity1 operated on for ruptured anterior circulation @entity439 , five @entity1 (11.6%) developed clinical signs of @entity2806 postoperatively. In four of those @entity1 , the diagnosis of " @entity2553 " was made with measurements of cerebral blood flow (133Xe inhalation or intravenous injection, 10-16 detectors, cerebral blood flow infinity). Treatment with induced @entity101 with @entity2647 was instituted. Hemodilution was instituted in one @entity1 ; the other three @entity1 already had hematocrits in the range of 33. Within 1 hour, the cerebral blood flow measurement was repeated to document the effect of treatment. The average pretreatment hemispherical blood flow on the operated side was 18.8 mL/100 g per minute, on the contralateral side 21.0 mL/100 g per minute. With treatment these flows increased to 30.8 and 35.8 mL/100 g per minute, respectively. There was also an immediate and obvious positive clinical effect in all @entity1 . The role of measurement of cerebral blood flow in the clinical management of @entity2553 is discussed. We stress the theoretical and practical advances of measurements of cerebral blood flow over cerebral angiography, especially in @entity772 @entity1 .
