Muscle Triggers as a Possible Source of XXXX in a Sub-group of Tension Type Headache Patients?

@entity1931 @entity158 @entity2701 @entity1867 @entity925
OBJECTIVES: @entity2701 ( @entity2701 ) is a common condition but the underlying etiology is not understood. Episodic @entity2701 may develop into chronic @entity2701 , and some possible triggers may be involved in generation and maintenance. Nociceptive generators and hyperexcitable @entity1867 in neck and shoulder regions may to some degree contribute to @entity2701 . The current paper highlights some of the possible triggers and associated @entity158 mechanisms involved in @entity2701 and discusses if inhibition of these possible triggers may provide new treatment options. RESULTS: This paper presents possible pathophysiological factors in @entity2701 , the role of @entity925 and how referred @entity158 from triggers can contribute to development and/or maintenance of sensitization. @entity158 patterns from trigger points and associated @entity1931 seem to be clinically important factors. Damping the nociceptive peripheral drive may not only reduce the number of @entity2701 attacks but may also prevent/delay the transition from episodic into more chronic @entity2701 . The role of muscle triggers in driving @entity2701 is debated as the pathogenesis of such triggers is not fully understood. Furthermore, inhibiting the drive from the triggers does not consistently modulate @entity2701 . DISCUSSION: Understanding the possible triggers in @entity2701 , @entity1931 , and @entity158 sensitization, may help to develop better management regimes and possibly prevent @entity2701 from developing into more chronic conditions. Currently, there is a striking difference between the clinical observational studies favoring the role of muscle triggers in @entity2701 and the intervention studies generally not supporting the role of muscle triggers in @entity2701 .
