[A new @entity65 : recurrent @entity1163 caused by lesions or @entity5 of the parapharyngeal space. Etiopathogenesis, clinical picture, differential diagnosis with @entity50 and XXXX . Therapy by intracranial resection of the 9th cranial nerve].

@entity1 @entity907 @entity5 @entity1163 @entity5587 @entity9 @entity158 @entity1167 @entity551 @entity50 @entity65 @entity9201
An intense vaso-vagal reaction characterizes all the reflex induced @entity65 . In these syndromes the vagal cardio-inhibitor effect on heart rate is more evident than the vasodilatation and @entity551 . The vasodepressor mechanism is uncommon even in @entity50 . We have studied 6 male @entity1 , age range 56-73 years (mean age: 64) with recurrent @entity907 . The following were always present during such episodes: generalized malaise, profound fatigue, pallor, cyanosis, @entity9201 , lack of peripheral pulses, severe @entity551 (BP) (systolic BP less than or equal to 50-60 mmHg or unrecordable), @entity1163 . The first diagnosis in our @entity1 was @entity50 , but, the clinical picture was quite different from classic @entity50 : triggering factors were not present, the vasovagal episodes were longer, the @entity1163 more frequent and severe, and the VVI pacing uneffective. Further investigations, including computerized axial tomography, showed--in all these @entity1 --a @entity5 originally localized in or near the parapharyngeal space. We think that the symptoms of our @entity1 can be attributed to parapharyngeal @entity5 and that the parapharyngeal space lesions are able to cause severe @entity907 and @entity1163 . The pathogenetic mechanism in this syndrome, due to neural irritation of the glossopharyngeal afferent fibres, is similar to the @entity1167 , but it obviously doesn't involve @entity158 -pathways since none of our @entity1 had @entity158 . Therefore, this syndrome differs from @entity5587 - @entity1167 in the presence of @entity5 and in the absence of @entity9 and initiating factors.(ABSTRACT TRUNCATED AT 250 WORDS)
