The prognostic value of evoked responses from primary somatosensory and auditory cortex in comatose XXXX .

@entity1 @entity131 @entity64 @entity198 @entity772 @entity1167 @entity12870
OBJECTIVE: To evaluate somatosensory and auditory primary cortices using somatosensory evoked potentials (SEPs) and middle latency auditory evoked potentials (MLAEPs) in the prognosis of @entity12870 in @entity772 @entity1 . METHODS: SEPs and MLAEPs were recorded in 131 severe @entity772 @entity1 . Latencies and amplitudes were measured. @entity772 had been caused by transient @entity1167 (n=49), @entity131 (n=22), @entity64 (n=45), complications of neurosurgery (n=12) and @entity198 (n=3). One month after the onset of @entity772 @entity1 were classified as awake, still @entity772 or dead. Three months after (M3), they were classified into one of the 5 categories of the Glasgow outcome scale (GOS). RESULTS: At M3, 41.2% were dead, 47.3% were conscious (GOS 3-5) and 11.5% had not recovered consciousness. None of the @entity1 in whom somatosensory N20 and auditory Pa were absent did @entity12870 and in the post-anoxic group, reduced cortical amplitude too was always associated with bad outcome. Conversely, N20 and Pa were present, respectively, in 33/69 and 34/69 @entity1 who did not recover. CONCLUSIONS: The prognostic value of SEPs and MLAEPs in @entity772 @entity1 depends on the cause of @entity772 . Measurement of response amplitudes is informative. Abolition of cortical SEPs and/or cortical MLAEPs precludes post-anoxic @entity772 @entity1 from returning to consciousness (100% specificity). In any case, the presence of short latency cortical somatosensory or auditory components is not a guarantee for @entity12870 . Late components should then be recorded.
