Renal involvement in XXXX : sonographic-pathologic correlation.

@entity1 @entity8 @entity66 @entity631 @entity791
Renal sonography was performed in 36 @entity1 with clinical and/or laboratory evidence of @entity791 , usually because of deteriorating renal function. In 15 @entity1 , histopathologic specimens were reviewed to characterize renal pathologic changes underlying the sonographic findings. Sonographic evaluation included determination of renal size and cortical echogenicity according to a standard grading system. Pathologic specimens were evaluated for tubular and @entity8 . Sonography showed either normal-sized or enlarged kidneys with grade I cortical echogenicity in 13 @entity1 (36%), grade II in three @entity1 (8%), and grade III in five @entity1 (14%). Fifteen @entity1 (42%) had normal renal echogenicity. In addition to focal segmental @entity631 the pathologic examination showed different degrees of @entity66 . Striking, irregularly dilated, infolded tubules with flattened epithelium and intratubular deposits of proteinaceous material, and sometimes cystlike formation involving the medulla and cortex, were seen in two @entity1 with @entity8 , and mild dilatation of the tubules was seen in four @entity1 with grade I disease. Moderate tubular dilatation was observed in one @entity1 with grade I disease. No significant @entity66 was seen in one @entity1 with grade I disease or in seven @entity1 with normal renal echogenicity. Although glomerular changes contribute to the increase in renal echogenicity, we postulate that the main factors responsible for the increased echogenicity in @entity8 are the striking @entity66 seen in these @entity1 .
