XXXX and pulmonary symptoms and signs in @entity12767 and their families in Los Angeles.

@entity3617 @entity66 @entity565 @entity12767 @entity1576 @entity1224 @entity393 @entity1355
Families of 338 male and 81 female @entity12767 ( @entity12767 ), including 280 wives, 144 daughters, and 81 sons, were examined for diseases resulting from @entity1355 . The workers were initially exposed to @entity1355 at least 20 years prior to the study date. Radiographic signs of @entity1355 (using standard criteria international Labor Office 1980) were found in 64% of 288 male @entity12767 and 21% of 71 female @entity12767 . After excluding those with any occupational exposure to @entity1355 , @entity1355 prevalence was 11% in wives, 8% in sons, and 2% in daughters. @entity66 prevalence in workers and in wives increased with the number of years from initial exposure. Male @entity12767 who had smoked had @entity1224 without @entity393 . Nonsmokers had normal pulmonary functions. In @entity12767 households prevalences of @entity1576 , wheezing on physical examination, and symptoms of @entity565 and chronic @entity3617 , exceeded those in the comparison (Michigan) population, even for the younger daughters and sons. These differences, and @entity1224 and distribution defects reported earlier were not explained by cigarette smoking or by @entity1355 exposure. Instead, they are tentatively ascribed to long-term exposure to ambient air pollution in Los Angeles.
