Neurocognition in XXXX .

@entity1 @entity73 @entity51 @entity161 @entity4207
@entity51 among @entity161 @entity1 have been consistently documented in research over the past 20 years and are reviewed in this chapter. Discussion of general abilities is presented as a background and is followed by analysis of functioning in specific cognitive domains. Overall intellectual deficits are indicated by results from both general intelligence tests and composite test battery scores. Within specific cognitive domains, effect size differences are noted in numerous areas, including attention, with indications that working memory is affected more severely than simple attention, likely due to inclusion of an executive component in such tasks. There is also evidence of slowed processing speed among @entity161 @entity1 , likely contributing to deficits in other domains which rely on rapid and efficient assimilation of information. @entity73 have been found on tests assessing set-shifting abilities, selective attention, and inhibition of inappropriate responses. @entity4207 have been demonstrated extensively, with some evidence that recall of verbal material is more affected than recall of visual information, and that recognition abilities are comparatively less impaired than recall for both modalities. Receptive and expressive language abilities are compromised in @entity161 @entity1 , as well as visual perceptual, constructional, and fine motor skills. Social cognition is an area of particular importance due to its relevance to functional outcome. Deficits in expression and recognition of facial and prosodic affect have been demonstrated, although subjective experience of emotion appears to be relatively well preserved. @entity51 described in this review appear to generally remain stable throughout adulthood, supporting neurodevelopmental, rather than neurodegenerative, models of the illness. Finally, @entity73 are increasingly used as endophenotypes, which is likely an important direction of future research.
