Hyperperfusion injury of the XXXX kidney in different glomerular diseases.

@entity1 @entity101 @entity6 @entity3630 @entity1011 @entity631
Investigations of biopsy material from @entity1 kidneys with different forms of @entity631 (n = 1,240) and with @entity6 (n = 406) performed in order to find changes caused by hyperperfusion of the kidney tissue gave the following results: (1) Hyperperfusion injury occurs in the different forms of @entity631 with varying frequency. It was rarely found in immunologically negative mesangioproliferative @entity631 . The highest incidence was found in @entity1 with @entity3630 was also found in kidneys with @entity6 . The frequency of this finding increased with the degree of the @entity6 changes. (3) The hyperperfusion injury was seen as a complication of @entity631 or @entity6 only when the @entity1 clinically had developed malignant @entity101 and when the serum @entity1011 level was elevated, a sign of compensated retention. (4) In @entity1 with @entity631 , the hyperperfusion changes occurred more frequently in males than in females. @entity6 was complicated by hyperperfusion injury with the same frequency in both sexes. (5) @entity1 with hyperperfusion changes of the kidneys always excrete large amounts of protein in the urine. (6) Hyperperfusion changes occur first in the juxtamedullary glomeruli. The intermediate glomeruli are affected later and the subcapsular glomeruli last.
