Presence of @entity3883 in XXXX .

@entity1 @entity3883 @entity712 @entity296 @entity82 @entity2686 @entity1555 @entity299 @entity204
BACKGROUND: @entity299 in @entity1 with @entity1555 ( @entity1555 ) is a leading cause of @entity204 . Cardiac resynchronization therapy (CRT) has been shown to dramatically decrease mortality in eligible adult population with @entity712 . We hypothesized that @entity3883 is present in @entity1555 @entity1 and that cardiovascular magnetic resonance (CMR) may predict CRT efficacy. METHODS: @entity1555 @entity1 (n = 236) were stratified into 4 groups based on age, diagnosis of @entity1555 , left ventricular (LV) ejection fraction (EF), and presence of @entity296 defined as positive late gadolinum enhancement (LGE) compared to normal controls (n = 77). Dyssynchrony indices were calculated based on timing of CMR derived circumferential strain (ecc). The calculated indices included cross-correlation delay (XCD), uniformity of strain (US), regional vector of variance (RVV), time to maximum strain (TTMS) and standard deviation (SD) of TTMS. Abnormal XCD value was defined as > normal + 2SD. US, RVV, TTMS and SD were calculated for @entity1 with abnormal XCD. RESULTS: There was overall low prevalence of circumferential dyssynchrony in the entire @entity1555 population; it increased to 17.1% for @entity1 with abnormal EF and to 31.2% in the most advanced stage (abnormal EF with @entity82 ). All but one @entity1555 @entity1 with @entity3883 exhibited normal QRS duration suggesting absence of electrical dyssynchrony. The calculated US and RVV values (0.91    0.09, 1.34    0.48) indicate disperse rather than @entity2686 . CONCLUSION: Mechanical dyssynchrony is frequent in @entity1 with end stage @entity1555 -associated @entity299 . It is associated with normal QRS complex as well as extensive lateral @entity82 . Based on these findings, it is unlikely that this @entity1 population will benefit from CRT.
