Effects of exercise on postexercise @entity67 coupling and pulsatile efficiency in @entity1 with XXXX .

@entity1 @entity308 @entity712 @entity392 @entity299 @entity67
BACKGROUND: A suboptimal @entity67 ( @entity67 ) interaction may have a prolonged @entity308 effect on the failing heart after functional reserves forced to their limits under stress conditions such as exercise. The continuation of excessive load in the postexercise period may be more important than the load during exercise, because the sum of postexercise periods generally exceeds exercise time itself. We sought that exercise-induced changes in postexercise @entity67 coupling and pulsatile efficiency in @entity1 with @entity712 ( @entity712 ). METHODS: Thirty consecutive @entity712 with reduced ejection fraction (EF) and thirty age-, sex- and peak VO2 -matched subjects with preserved EF were enrolled. Pre- and postexercise echocardiographic and tonometric measurements were taken to calculate left ventricular and arterial elastances, arterial compliance and wave reflections, and steady and pulsatile power. RESULTS: @entity67 coupling significantly deteriorated in @entity712 group (from 1  50      0  47 to 2  00      0  75  mmHg/mL, P  <  0  01), but control group maintained basal favourable coupling status after exercise (from 1  04      0  29 to 1  03      0  24  mmHg/mL, P  =  0  77). Pulsatile percentage of total power significantly increased with exercise in @entity712 group, whereas it showed a significant decrease in control group. The change in pulsatile power fraction was correlated with the change in augmentation pressure (r  =  0  41,     =  3  00, P  <  0  01) and inversely correlated with the change in total arterial compliance (r  =  -0  29,     =  -8  52, P  =  0  02). CONCLUSION: Our data indicate that exercise-induced @entity67 decoupling and @entity392 extend into postexercise phase in @entity1 with @entity299 . The exact duration of these derangements requires further studies.
