[The role of " XXXX alleles" in the pathogenesis of polyglobulia after renal transplantation].

@entity1 @entity205 @entity500 @entity9595
43 kidney recipients were followed up for 12 months after renal transplantation. In 13 @entity1 treated with @entity205 for 6 months polyglobulia was found, which subsided after conversion of immunosuppressive therapy. A highly significant correlation was found between the presence of the antigen A3 and the polyglobulia in kidney recipients treated by @entity205 . The serum ferritin level was not elevated in kidney recipients who were carriers of the " @entity9595 alleles". Results obtained in this study seem to justify the following conclusions: 1. Kidney recipients who are carriers of " @entity9595 alleles" show a threefold higher risk of posttransplant polyglobulia. 2. Presence of " @entity9595 alleles" is not an absolute predictor of polyglobulia in @entity1 after kidney transplantation. 3. Both etiology and pathogenesis of posttransplant polyglobulia do not seem to be homogeneous. @entity205 , in contrast to @entity500 , seems to have a promoting effect on the development of posttransplant polyglobulia. Therefore treatment of kidney transplant @entity1 with @entity205 , who are carriers of " @entity9595 alleles", needs careful monitoring.
