@entity3190 and XXXX . Cause and effect.

@entity43 @entity1 @entity3610 @entity66 @entity1640 @entity3428 @entity15773 @entity3190 @entity342
The evidence that supports the hypothesis that @entity3190 causes @entity43 was reviewed. The most dramatic clinical expression of the association between @entity3190 and @entity3428 is the @entity15773 . In the @entity15773 , severe insulin resistance results in a @entity3190 that stimulates ovarian @entity342 production. Acanthosis nigricans, a dermatologic manifestation of severe insulin resistance, is an @entity66 . Hyperandrogenic (HA) @entity1 can be divided into two major groups: insulin-resistant (HA-IR) and non-insulin-resistant (HA-nIR). HA-nIR @entity1 have markedly elevated LH, often have minimally elevated serum prolactin, and have @entity3610 . HA-IR @entity1 have minimally elevated LH, markedly elevated plasma insulin, and stromal hyperthecosis. @entity1 with HA-nIR probably have a primary @entity1640 as the cause of their @entity3428 . @entity1 with HA-IR probably have a primary metabolic abnormality as the cause of their @entity3428 . The relationship between @entity3190 and @entity3428 is one example of the complex interrelationships that exist between central metabolism and reproduction.
