Trace elements and XXXX .

@entity1 @entity1423 @entity4259 @entity1674 @entity219 @entity3407 @entity920 @entity299 @entity413 @entity65 @entity165
Evidence linking marginal intakes of the trace elements, @entity3407 , @entity920 , @entity219 and @entity4259 , with abnormal lipid metabolism and ultimately @entity65 is accumulating from both animal and @entity1 studies. @entity3407 supplementation of normal adult @entity1 , as well as diabetics, has been reported to increase high density lipoprotein @entity165 and decrease @entity1674 and total @entity165 . Subjects with the highest total @entity165 and @entity1674 usually respond the most to supplemental @entity3407 . Improvements in lipid metabolism, as well as those in @entity413 metabolism, appear to be related to improvements in insulin efficiency due likely to increased receptor number. Animal studies also indicate that improvements in serum @entity165 , aortic lipids and plaque formations due to supplemental @entity3407 are associated with decreased circulating insulin. Insufficient dietary @entity920 also leads to elevated lipid levels and @entity299 . Animal studies indicate an obvious degradation of the heart muscles. @entity219 appears to function in @entity65 primarily via its antagonism with @entity920 . @entity4259 may also affect @entity65 since @entity4259 is postulated to be involved in @entity1423 . These data demonstrate that the trace elements, @entity3407 , @entity920 , and @entity4259 , have beneficial effects on risk factors associated with @entity65 suggesting that a decreased risk of @entity65 may be achieved by adequate intake of trace elements.
