XXXX and @entity239 .

@entity1 @entity239 @entity82 @entity294 @entity843 @entity1373 @entity344 @entity1960 @entity490 @entity1577
OBJECTIVE: To describe our experience with @entity1577 ( @entity1577 )-associated @entity1960 and @entity239 , and to assess treatment strategies. STUDY DESIGN: We reviewed hospital charts and autopsy reports of all University of Minnesota @entity1 with @entity1577 between 1975 and August 1994. We identified six @entity1 with colonopathy and compared them with a cohort of 79 @entity1 with @entity1577 in the same age range and seen during the same period. RESULTS: All @entity1 with colonopathy had bloody @entity294 ; five of the six had @entity490 . Stool frequency and related symptoms distinguished the @entity1 with colonopathy from the cohort population. All took a higher median dose of pancreatic enzymes than the cohort population during the 3 months preceding the onset of symptoms (p < 0.002). For all six @entity1 , barium studies revealed loss of haustration, and shortening and diffuse narrowing of the colonic lumen with relative rectal sparing. The distal ileal mucosa was irregular in four @entity1 . A histopathologic study reveal @entity82 , and focal acute cryptitis in all six @entity1 . Other features included @entity843 (2/6) and nodular regenerative @entity1373 of the liver (1/6). One @entity1 continues to have symptoms, three had subtotal colectomy, and the condition of two improved after a regimen including a low-fat diet, withholding of pancreatic enzymes, and supplemental parenteral nutrition was initiated. CONCLUSIONS: @entity239 represents a newly recognized @entity344 of @entity1577 . Affected @entity1 have taken larger doses of pancreatic enzymes than similar @entity1 with @entity1577 , and have bloody @entity294 . We developed a medical protocol that may avoid surgical resection of the colon in some of these @entity1 .
