The ventral portion of the anterior pretectal nucleus controls descending mechanisms that initiate @entity9 in XXXX .

@entity5019 @entity128 @entity56 @entity8638 @entity35 @entity282 @entity9
UNASSIGNED: Stimulating the dorsal anterior pretectal nucleus (dAPtN) in @entity35 is more effective than stimulating the ventral APtN (vAPtN) at reducing tail-flick latency, whereas stimulation of the vAPtN is more effective at reducing @entity282 behavior. This study examines whether the cell lesion by injecting N-methyl-D-aspartate into the dAPtN or vAPtN changes the withdrawal threshold of a @entity35 hind paw during different phases of the tactile @entity56 induced by a @entity8638 ( @entity8638 ) of the contralateral sciatic nerve. The number of @entity5019 immunoreactive cells in the APtN was also evaluated. The @entity35 whose vAPtN was lesioned 2 days before @entity8638 had more intense tactile @entity56 2 days after @entity8638 than that of the control, but the groups were not different 7 days after the @entity8638 . The @entity35 whose vAPtN was lesioned 5 days after @entity8638 had WTs that did not significantly differ 7 days after the @entity8638 . The tactile @entity56 of the @entity35 whose dAPtN was lesioned 2 days before or 5 days after @entity8638 was not different from that of the control on the 2(nd) and 7(th) days after the @entity8638 . The number of @entity5019 immunoreactive cells in the vAPtN and dAPtN increased 2 days after @entity8638 , but did not differ from that in the control 7 days after @entity8638 . We conclude that vAPtN and dAPtN cells are activated by @entity128 ; the vAPtN exerts inhibitory control of the initial phase of @entity9 whereas the dAPtN does not appear to exert inhibitory role in neuropathic processing. This article is protected by copyright. All rights reserved.
