XXXX metabolism and plasma protein turnover in @entity30 .

@entity1 @entity2226 @entity2228 @entity294 @entity344 @entity1960 @entity6741 @entity3645 @entity30
Postprandial duodenal @entity3645 , intestinal protein loss, and albumin and IgG turnover were studied in 19 non-operated @entity1 with @entity30 . A lesion of the terminal ileum was present in 18 of 19 @entity1 , either alone or associated with @entity1960 . Identical @entity6741 studies were made in a control group of 20 @entity1 with chronic @entity294 of undetermined origin. Duodenal @entity6741 concentration was decreased in 9 of 19 @entity1 with @entity30 , and in 5 of 20 @entity1 with unexplained @entity294 . The @entity2228 / @entity2226 -ratio was increased in 8 of 17 @entity1 with @entity30 , but in only one of the 20 control @entity1 . Abnormal intestinal protein loss was present in 13 of 14 @entity1 with @entity30 . The fractional catabolic rate of albumin and IgG was increased in all 17 cases of @entity30 studied, except the @entity1 with no protein loss. A statistically significant and positive correlation was observed between @entity2228 / @entity2226 -ratio and fractional catabolic rate of both albumin and IgG. No @entity1 with @entity30 harboured an abnormal bacterial flora in the proximal small intestine. It is concluded that, in the absence of abnormal bacterial flora in the proximal jejunum, the @entity2228 / @entity2226 -ratio is more valuable as an indicator of terminal ileopathy than postprandial duodenal @entity6741 concentration in nonoperated @entity1 with @entity30 . Abnormal intestinal protein loss and increased catabolic rate of albumin and IgG are practically always present in active @entity30 and are strong evidence of an organic @entity344 in @entity1 with normal radiographic findings.
